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Wei Shi Liang
Intensive Care Unit
About me
Graduated from Shanxi Medical University with a degree in Clinical Medicine in 2006, and has been working in the field of Critical Care Medicine ever since.
Proficient in diseases
Treatment of severe infections, ARDS, severe trauma, MODS, and other diseases.
Voices
What kind of urine occurs with hyperkalemia?
Primary hyperkalemia often coincides with metabolic acidosis, and in hyperkalemia-induced metabolic acidosis, paradoxical alkaline urine can occur. Once hyperkalemia occurs, it primarily affects the conduction of the heart and neuromuscular system. Typical clinical manifestations include severe bradycardia, atrioventricular conduction block, and even sinus arrest. In mild hyperkalemia, the electrocardiogram shows peaked T-waves; as potassium levels continue to rise, the PR interval prolongs, T-waves disappear, QRS complex widens, and ultimately, cardiac arrest occurs. Immediate treatment should be administered upon diagnosis to promote the excretion of potassium, maximizing the renal excretion capacity with diuretics. If drug-induced potassium excretion does not normalize levels and serum potassium exceeds 6.5 mmol/L, hemodialysis may be necessary. Additionally, some drugs can be used to shift potassium into the cells and protect cardiac function. (The use of any medication should be under the guidance of a doctor.)
Causes of Bronchial Asthma
The most common cause of bronchial asthma is inhalants, which are mainly found in daily life, such as dust mites, pollen, fungi, and some irritating and toxic gases, all of which may irritate the airways and cause asthma. Infections are closely related to the occurrence of asthma, and respiratory infections such as viruses, bacteria, and mycoplasma can all trigger asthma. Additionally, certain specific foods, such as fish, shrimp, crab, milk, and eggs, may cause asthma in certain groups of people. Factors such as the patient's mental state, changes in the climate, and intense exercise are also possible causes of bronchial asthma. These are the most common causes of bronchial asthma onset.
Causes of acute heart failure include
Most patients with acute heart failure have a history of heart disease, and the common causes mainly include: 1. Acute myocardial necrosis or damage, such as acute coronary syndrome, peripartum cardiomyopathy, and myocardial damage caused by drugs or toxins, including sepsis-induced myocardial damage. 2. Acute exacerbation of chronic heart failure due to infection or other stressful factors. 3. Acute hemodynamic changes, mainly including conditions like cardiac tamponade, hypertensive crisis, aortic dissection, and acute valvular regurgitation, all of which can lead to acute heart failure.
Causes of hypokalemia
Potassium is one of the essential electrolytes necessary for life. Its physiological functions mainly include maintaining cellular metabolism, regulating osmotic pressure and acid-base balance, and preserving cell stress response, etc. Daily potassium intake is about 100 millimoles, with 90% excreted through the kidneys and the remainder through the gastrointestinal tract. Common causes of hypokalemia include reduced intake, such as long-term inability to eat without timely potassium supplementation. Even though potassium intake decreases, the kidneys continue to excrete potassium, leading to potassium loss. The second cause is increased excretion, which includes losses through the gastrointestinal tract and the kidneys, both of which can lead to hypokalemia. The third cause is the movement of potassium from outside to inside the cells, which can occur during metabolic alkalosis or when glucose and insulin are administered, promoting the transfer of potassium ions into the cells, resulting in hypokalemia.
Causes of Acute Heart Failure
The main causes of acute heart failure include coronary heart disease, valvular disease, hypertension, and cardiomyopathies, such as toxic cardiomyopathy or hypothyroidism-related cardiomyopathy, as well as idiopathic cardiomyopathy. Myocarditis and arrhythmia-related causes can also lead to heart failure, but there are often triggers present clinically. Common triggers include poor treatment compliance, arrhythmias, anemia, infections, myocardial ischemia, excessive fluid intake, poor dietary control, and increased cardiac output, such as during strenuous activity and pregnancy, which can lead to increased cardiac output and cause heart failure. Conditions such as excessive fluid volume, hypertension, hyperthyroidism, and pulmonary embolism can also trigger heart failure.
Pulmonary Embolism Interventional Treatment Methods
Interventional treatment for pulmonary embolism is mainly used for large pulmonary embolisms in the main trunk or major branches of the pulmonary artery. It is applicable in the following scenarios: contraindications to thrombolysis and anticoagulation therapy, inefficacy after thrombolysis or aggressive medical treatment, or lack of surgical conditions. Interventional therapy for pulmonary embolism can involve removing the embolus or breaking it into fragments, allowing it to move to the distal pulmonary arteries, thereby opening the central pulmonary arteries, rapidly reducing pulmonary artery resistance, significantly increasing total pulmonary blood flow, improving cardiopulmonary hemodynamics, and right ventricular function. The treatment involves catheter fragmentation and suction of large clots in the pulmonary artery or performing balloon angioplasty, and it also enables local administration of small-dose thrombolysis. These are the primary methods of interventional treatment for pulmonary embolism.
Causes of Hypokalemia
Common causes of hypokalemia include insufficient intake or prolonged inability to eat without intravenous supplementation. In such cases, while intake of potassium decreases, the kidneys continue to excrete potassium, leading to a loss of potassium in the blood. Additionally, increased excretion can cause hypokalemia, including losses from the gastrointestinal tract such as vomiting, diarrhea, and continuous gastrointestinal decompression, which results in a loss of digestive fluids rich in potassium. Potassium loss through the kidneys from prolonged use of potassium-wasting diuretics or during the polyuric phase of acute renal failure can also lead to hypokalemia. Furthermore, the shift of potassium from outside to inside the cells can cause hypokalemia.
Can people with hypokalemia smoke?
Hypokalemia is not directly related to smoking. However, once hypokalemia occurs, there is definitely an underlying disease. In the case that the primary disease is not controlled, it is advisable to avoid smoking. Potassium is an essential electrolyte for life, and its physiological functions mainly include maintaining cellular metabolism, regulating osmotic pressure, acid-base balance, and maintaining cell stress functions. Once hypokalemia occurs, active treatment should be implemented, primarily addressing the primary disease, symptomatic treatment with potassium supplementation, and avoiding the occurrence of hyperkalemia. The principle of potassium supplementation is that for mild hypokalemia without clinical manifestations, oral potassium should be given; in cases of severe hypokalemia, intravenous potassium supplementation should be administered immediately. Intravenous potassium should ideally not use peripheral veins but establish a central vein, and the speed of potassium supplementation and the monitoring of potassium levels should be controlled.
Hypokalemia belongs to the department of nephrology.
Hypokalemia is seen in various clinical departments and can affect the nervous system, muscles, heart, digestive system, kidneys, as well as carbohydrate metabolism and acid-base balance. If hypokalemia occurs, it is important to first identify the primary disease and treat it specifically in the corresponding department. In cases of severe hypokalemia, patients should be admitted to the intensive care unit. Treatment involves addressing the primary disease and promptly supplementing potassium. Severe hypokalemia, especially if accompanied by arrhythmias or muscle paralysis, requires immediate potassium supplementation. Potassium deficiency within cells recovers slowly; treatment may take four to six days to gradually reach a balance. Additionally, it is important to timely correct other electrolyte imbalances. The specific department to which the patient is admitted mainly depends on the primary disease, but in cases of very severe conditions, potassium supplementation should be managed in the intensive care unit.
The earliest clinical symptoms of respiratory failure
The clinical symptoms of respiratory failure mainly manifest as hypoxemia and carbon dioxide retention. Looking at their clinical manifestations, mild hypoxia may not show any clinical symptoms. As the severity of the disease increases, there can be signs of increased respiratory drive, such as rapid breathing or difficulty breathing, along with symptoms of sympathetic nervous system activation, such as anxiety and sweating. Hypoxemia can cause dilation of peripheral arteries and constriction of veins, leading to an increased heart rate, or even severe arrhythmias. Furthermore, considering the clinical manifestations of carbon dioxide retention, they are directly related to the levels of carbon dioxide in the body, mainly depending on the rate of occurrence. Clinically, this primarily affects myocardial contractility, the contractile ability of respiratory muscles, and increases in intracranial blood flow. In mild to moderate cases, it can stimulate the respiratory center causing increased and shallow breathing, but in severe cases, it may suppress the respiratory center.