Albinism in infants is caused by a deficiency or dysfunction of tyrosinase, leading to a lack of melanin or a synthesis disorder in the skin and its appendages. This inherited form of albinism results in the retina lacking pigment, with the iris and pupils appearing pale pink, and sensitivity to light. The skin, mucous membranes, hair, and other body hair are white or yellowish-white. Albinism is a familial hereditary disease, inherited as an autosomal recessive trait, often occurring in populations with a high rate of consanguineous marriages. In the genetic pedigree of albinism, both parents carry the albinism gene; carriers themselves do not show symptoms but can pass the causative gene to their children, who then manifest the disease.