The key to curing iron deficiency anemia

Written by He Li Fang
Hematology
Updated on September 13, 2024
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Iron deficiency anemia, a common and frequently occurring disease in the department of hematology, is not difficult to diagnose and treat. The key to treatment lies in addressing the cause of the iron deficiency, and it is essential to eliminate the underlying causes as much as possible. Using iron supplements alone can only restore blood counts; neglecting the primary disease will not lead to a complete cure of the anemia and can easily lead to recurrence. Treatment consists of two phases: first, the supplementation with iron supplements. After taking iron supplements for three to four days, an increase in reticulocytes will occur, reaching a peak around seven days, and hemoglobin will significantly rise after two weeks of medication, returning to normal levels after one to two months. However, after hemoglobin levels normalize, some patients may stop taking the medication. It is important to note that continuing to take iron supplements for three months is necessary to replenish the body's iron stores completely.

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Written by He Li Fang
Hematology
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Characteristics of iron deficiency anemia blood picture

The clinical manifestations of iron deficiency anemia are due to the specific characteristics of anemia caused by iron deficiency and the underlying disease causing the iron deficiency. Its hematological characteristics present as typical microcytic hypochromic anemia. What does this concept mean? It refers to the values in a complete blood count where MCV is less than 80 femtoliters, MCH is less than 27 picograms, and MCHC is less than 30%. MCV refers to the average red blood cell volume, MCH refers to the mean corpuscular hemoglobin concentration, and MCHC refers to the mean corpuscular hemoglobin concentration of red blood cells. The extent of changes in red blood cell indices correlates with the duration and severity of anemia. The blood smear typically shows pale-stained red blood cells with an enlarged central pallor and varying sizes. Platelet count often tends to be high when there is concurrent bleeding and generally tends to be low in infants and children. White blood cell count is usually normal or slightly reduced, with normal differential. These are the hematological characteristics of iron deficiency anemia.

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Written by He Li Fang
Hematology
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The key to curing iron deficiency anemia

Iron deficiency anemia, a common and frequently occurring disease in the department of hematology, is not difficult to diagnose and treat. The key to treatment lies in addressing the cause of the iron deficiency, and it is essential to eliminate the underlying causes as much as possible. Using iron supplements alone can only restore blood counts; neglecting the primary disease will not lead to a complete cure of the anemia and can easily lead to recurrence. Treatment consists of two phases: first, the supplementation with iron supplements. After taking iron supplements for three to four days, an increase in reticulocytes will occur, reaching a peak around seven days, and hemoglobin will significantly rise after two weeks of medication, returning to normal levels after one to two months. However, after hemoglobin levels normalize, some patients may stop taking the medication. It is important to note that continuing to take iron supplements for three months is necessary to replenish the body's iron stores completely.

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Written by He Li Fang
Hematology
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How long does iron deficiency anemia need to be treated?

Patients with iron deficiency anemia typically need several months of iron supplementation treatment. The specific reason is that after taking iron supplements, patients' subjective symptoms can recover quickly. The count of reticulocytes generally begins to rise within three to four days after starting the medication, reaching a peak around seven days. Hemoglobin levels notably increase within two weeks of treatment, and generally return to normal around two months. After correcting anemia, it is necessary to continue treatment for at least another three months because the body's iron stores need further supplementation. It is necessary to restore serum ferritin levels to fifty micrograms per liter to replenish these stores fully. This is required to achieve a true cure of iron deficiency anemia, as without this, patients are prone to relapse. Therefore, regular and long-term effective use of iron supplements is an essential basis for ensuring the treatment effectiveness of iron deficiency anemia.

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Written by He Li Fang
Hematology
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Diet for Iron Deficiency Anemia

Patients with iron-deficiency anemia need to undergo pharmacological treatment with oral or injective iron supplements. In addition, patient health education should be enhanced to guide them in following a scientific and reasonable diet. For example, patients who are picky eaters or have insufficient intake should change their dietary structure and lifestyle in order to increase iron intake and bioavailability. This ensures that all key nutrients involved in the production of red blood cells are sufficient. Moreover, it is necessary to increase the intake of iron-rich foods, mainly including animal liver, blood, as well as soybeans, black fungus, egg yolks, brown sugar, lean meat, sheep kidneys, dried fruits, and sesame paste.

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Written by Zhang Xiao Le
Hematology
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Does iron deficiency anemia cause a yellow complexion?

No matter what type of anemia, it manifests as a decrease in hemoglobin. Red blood cells are the main cells that carry oxygen, so patients with anemia will have signs of ischemia and hypoxia to some extent, and anemia manifests as pallor in the skin and mucous membranes. Since Chinese are of the yellow race, the manifestation of anemia in Chinese patients often appears as a yellowish complexion. Therefore, patients with iron deficiency anemia can have a yellowish complexion. Of course, if there is obvious yellowing of the skin and sclera in addition to the yellowish complexion, further tests such as hemolysis indicators are needed to rule out hemolytic anemia, as patients with hemolytic anemia often have concurrent jaundice.