Can rickets be cured?

Written by Li Jiao Yan
Neonatology
Updated on January 04, 2025
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Rickets is a chronic nutritional disease caused by vitamin D deficiency that leads to disturbed calcium and phosphorus metabolism in the body, causing abnormal skeletal development. If a square skull is observed, it indicates that the child's disease has entered an active phase. Generally, in the early stages, there is an enlargement of the fontanelle or a delay in the closure of the fontanelle. By seven to eight months, a square skull can appear, primarily characterized by outward bulging centered on the frontal and parietal bones. At this time, it is generally necessary to administer a sufficient dose of vitamin D and encourage appropriate outdoor activities. Depending on the child's condition, it might be appropriate to supplement with calcium as well. Gradually, this should be curable; however, if there are other special causes for the abnormal vitamin D levels, such as severe liver or kidney disease or some new genetic diseases causing square skull, the treatment might be more challenging.

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Written by Li Jiao Yan
Neonatology
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Can rickets be cured?

Can rickets be cured? Rickets is a type of nutritional disease caused by a deficiency of vitamin D, which disrupts the body's calcium and phosphorus metabolism and leads to abnormal bone development. The common form is nutritional vitamin D deficiency rickets. This type of disease is self-limiting and can be cured. Once children engage in sufficient outdoor activities and supplement their vitamin D levels, they can be completely cured. There are also some special cases, such as severe liver and kidney diseases caused by vitamin D deficiency, and some genetic diseases. These diseases require treatment to promote the absorption of vitamin D. However, for genetic diseases, treating rickets with vitamin D is ineffective, and these cases are more difficult to treat. For liver and kidney diseases, if the condition can be effectively controlled, they can also be cured.

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Written by Liu Li
Pediatrics
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What foods should not be eaten with rickets?

Patients with rickets should not eat some cereal foods, such as millet, rice, wheat, corn, and other foods. Cereal foods also contain vitamin D and calcium, and eating too much of these foods can exacerbate the disease. Patients with rickets should eat more light, low-fat, and easily digestible foods. They should control the intake of high-fat content daily and avoid eating stimulating and spicy foods. At the same time, they should increase their intake of vitamins, encourage the child to eat more fruits and vegetables to ensure an adequate supply of vitamins, and patients can also exercise more. More physical activity can help boost the immune system.

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Written by Yao Li Qin
Pediatrics
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Treatment of Rickets

Rickets is a nutritional metabolic disease caused by a lack of vitamin D, which also involves changes in the bones. Once rickets is definitively diagnosed, it is essential to start aggressive treatment. Treatment should be carried out under the guidance of a hospital doctor with high doses of vitamin D, while also appropriately supplementing the child with calcium. It's necessary to regularly draw blood to test for vitamin D levels and to monitor the improvement of various indicators in the child. When vitamin D levels have fully normalized, there will no longer be a need to administer high doses of vitamin D to the child, and the dosage can be adjusted to the physiological requirement. Additionally, it is crucial to increase the child’s outdoor activities and exposure to sunlight, as this is very helpful for the treatment and recovery of rickets. (For specific medication use, please follow the guidance of a doctor and do not self-medicate.)

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Written by Li Jiao Yan
Neonatology
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Early manifestations of rickets

Rickets is a disease caused by a lack of vitamin D, which leads to a disorder in calcium and phosphorus metabolism, resulting in abnormal bone development. It primarily appears in the early stages in infants within six months old, and particularly within three months after birth. The initial presentation is an increase in nervous excitability, such as fussiness and unease in children, symptoms like excessive internal heat and sweating, especially more sweat on the head. Continuous sweating can lead to significant baldness at the back of the head. Sometimes, children may exhibit constant head shaking, but these are not very specific symptoms. Early detection involves decreased blood calcium and phosphorus levels, increased parathyroid hormone levels, and decreased levels of related markers in blood tests. Early bone X-rays appear normal, so initial symptoms are not so typical. Therefore, it is recommended that infants be taken to a pediatrician for a physical examination as soon as possible, where their developmental status can be assessed and rickets can be diagnosed. If it is early-stage vitamin D deficiency rickets, timely supplementation of vitamin D levels can be an effective cure.

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Written by Li Jiao Yan
Neonatology
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Rickets symptoms

Rickets, also known as nutritional vitamin D deficiency rickets, is a chronic nutritional disease in children caused by inadequate vitamin D in their bodies, leading to disorders in calcium and phosphorus metabolism and characterized by changes in the growth plates. Its manifestation varies with different ages. In infants under six months, especially those under three months, symptoms often include increased nervous excitability, such as being easily irritable, restless, sweating, and shaking their heads due to scalp irritation. As the condition progresses in children under six months, the primary changes are seen in the skull; the edges of the anterior fontanelle are softer, and the skull is thinner. After six months, the softening of the skull disappears, but there can be ping-pong ball-like changes around the skull, leading to a box-shaped head from seven to eight months. Gradually, beading changes form, most notably around the 7th to 10th ribs. Around the age of one, children can develop a pigeon chest deformity, and in severe cases of rickets, a horizontal depression forms at the lower edge of the thoracic cage, known as the costal groove or Harrison's groove. Due to bone softening and muscle and joint laxity, when the child begins to stand and walk, the legs may become bow-legged or X-shaped, and in severe cases, can develop into 'K'-shaped leg deformities. Once the child starts to sit and stand, general ligament laxity can cause spinal deformities. Severe hypophosphatemia leads to muscle carbohydrate metabolism disorder, resulting in overall muscle laxity, decreased muscle tone, and subsequently reduced muscle strength.