What foods should not be eaten with rickets?

Written by Liu Li
Pediatrics
Updated on December 05, 2024
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Patients with rickets should not eat some cereal foods, such as millet, rice, wheat, corn, and other foods. Cereal foods also contain vitamin D and calcium, and eating too much of these foods can exacerbate the disease. Patients with rickets should eat more light, low-fat, and easily digestible foods. They should control the intake of high-fat content daily and avoid eating stimulating and spicy foods. At the same time, they should increase their intake of vitamins, encourage the child to eat more fruits and vegetables to ensure an adequate supply of vitamins, and patients can also exercise more. More physical activity can help boost the immune system.

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Written by Li Jiao Yan
Neonatology
1min 22sec home-news-image

Can rickets be treated?

Rickets is a disorder caused by a deficiency of vitamin D, leading to impaired calcium and phosphorus absorption, which then results in abnormal skeletal development. It is a chronic nutritional disease. Generally, the rickets commonly mentioned is due to nutritional vitamin D deficiency, which is treatable. Typically, encouraging children to engage in appropriate physical activities while supplementing with an adequate level of vitamin D can effectively control the disease, and it can even be cured. However, if there are special circumstances, such as significant liver abnormalities, renal function abnormalities, renal failure, or abnormalities in the thyroid or parathyroid glands, including parathyroid tumors, or congenital hereditary diseases, the treatment needs to be based on the cause in order to effectively control the disease and promote normal calcium and phosphorus metabolism in the body. Congenital hereditary diseases generally make treatment more challenging. Therefore, it is crucial for children with rickets to seek timely medical examination at a hospital. If it is a common case of nutritional vitamin D deficiency, parents should supplement vitamin D promptly according to the doctor's advice for treatment.

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Written by Wang Xiao Jing
Pediatrics
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How to prevent skeletal deformities in rickets

Rickets is mainly caused by a deficiency of vitamin D. Therefore, within half a month after a child's birth, a preventive dose of oral vitamin D should be administered. Continue breastfeeding, allow the baby to get plenty of sunlight when the weather is warm, and timely introduce calcium-rich supplementary foods. In the early and active stages of rickets, appropriate supplementation with calcium and vitamin D can prevent the occurrence of skeletal deformities caused by the disease.

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Written by Li Jiao Yan
Neonatology
1min 58sec home-news-image

Rickets symptoms

Rickets, also known as nutritional vitamin D deficiency rickets, is a chronic nutritional disease in children caused by inadequate vitamin D in their bodies, leading to disorders in calcium and phosphorus metabolism and characterized by changes in the growth plates. Its manifestation varies with different ages. In infants under six months, especially those under three months, symptoms often include increased nervous excitability, such as being easily irritable, restless, sweating, and shaking their heads due to scalp irritation. As the condition progresses in children under six months, the primary changes are seen in the skull; the edges of the anterior fontanelle are softer, and the skull is thinner. After six months, the softening of the skull disappears, but there can be ping-pong ball-like changes around the skull, leading to a box-shaped head from seven to eight months. Gradually, beading changes form, most notably around the 7th to 10th ribs. Around the age of one, children can develop a pigeon chest deformity, and in severe cases of rickets, a horizontal depression forms at the lower edge of the thoracic cage, known as the costal groove or Harrison's groove. Due to bone softening and muscle and joint laxity, when the child begins to stand and walk, the legs may become bow-legged or X-shaped, and in severe cases, can develop into 'K'-shaped leg deformities. Once the child starts to sit and stand, general ligament laxity can cause spinal deformities. Severe hypophosphatemia leads to muscle carbohydrate metabolism disorder, resulting in overall muscle laxity, decreased muscle tone, and subsequently reduced muscle strength.

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Written by Li Jiao Yan
Neonatology
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What department should I go to for rickets?

Rickets, fully known as nutritional vitamin D deficiency rickets, is a chronic nutritional disease characterized by skeletal lesions in children caused by insufficient vitamin D leading to disturbances in calcium and phosphorus metabolism. In the early stages of rickets, the main issues are disturbances in calcium and phosphorus metabolism and low levels of vitamin D. At this time, one can consult the endocrinology department to check the child's trace elements. If sequelae have already formed, such as obvious pigeon chest or developmental deformities of the lower limbs bones, then it would be appropriate to consult an orthopedic department for corrective treatment.

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Written by Yan Xin Liang
Pediatrics
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Does rickets have a hereditary component?

Rickets, also known as vitamin D deficiency rickets, is primarily a chronic nutritional disease characterized by skeletal lesions, caused by insufficient vitamin D in infants, children, or adolescents, which leads to disturbances in calcium and phosphorus metabolism. The main feature of this disease is the incomplete calcification of the growth plates and bone tissue in the growing long bones, due to the lack of vitamin D causing incomplete calcification of mature bones. This disease is most common in children under two years old. Its causes may be related to insufficient intake of vitamin D or insufficient sunlight exposure, as well as rapid growth. Additionally, other diseases may influence its development. Therefore, this disease is generally not closely related to genetics.