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Osteophyte
Can people with bone hyperplasia drink alcohol?
Bone hyperplasia is a protective response caused by the bones at joint areas being subjected to long-term muscle traction and wear. Bone hyperplasia itself is not a disease, nor does it affect drinking alcohol. Drinking alcohol has neither therapeutic effects nor does it cause pain related to bone hyperplasia, so whether patients with bone hyperplasia drink alcohol is not very significant. For the treatment of bone hyperplasia, the most important things are rest, reducing heavy labor, and avoiding heavy physical work or extensive sports activities. Medications can include oral anti-inflammatory pain relievers, blood circulation-promoting drugs, and cartilage-protecting drugs for symptomatic treatment. (Specific medications should be taken under the guidance of a physician.)
Cervical vertebral hyperplasia surgical methods
In clinical practice, for patients with severe cervical spondylosis, such as those showing clear compression of the spinal nerves, exhibiting numbness, weakness, and pain in one or both arms, instability in walking, a sensation of stepping on cotton, or even abnormal urination and defecation, it is necessary to actively pursue surgical treatment. The surgery usually requires general anesthesia and involves the removal of the intervertebral disc, enlargement and decompression of the spinal canal, and vertebral interbody fusion with internal fixation. This should suffice. After the surgery, it is necessary to wear a neck brace for protection for about a month and a half, after which the neck brace can be removed to allow for appropriate activities.
Causes of lumbar vertebral hyperostosis
The first reason is aging of the human body. As the body ages, the lumbar spine inevitably exhibits degenerative changes due to wear and tear from movement, and the majority of people over 60 show the formation of bone spurs in X-ray images. The second reason is injury caused by overexertion. Overexertion, due to excessive workload and excessive activity, aggravates the degeneration of the lumbar spine, leading to the formation of bone spurs. The third reason is a history of trauma. Trauma to the lumbar area during one's youth can lead to bone proliferation in the lumbar spine in middle and old age. The fourth reason is improper posture. Bone proliferation in the lumbar spine in young patients is mainly due to maintaining the same posture for long periods, improper posture, and sleeping on soft beds, all of which can lead to bone proliferation over time. These are the main reasons for bone proliferation in the lumbar spine currently observed in clinical settings.
Is bone hyperplasia easy to treat?
The treatment of bone hyperplasia is relatively complex. Because bone hyperplasia will exist for a long time, most treatments are mainly aimed at improving the clinical symptoms of patients. Although symptoms improve after active cooperation with the treatment, the pathological basis of bone hyperplasia always exists. Later, in the case of increased numbers or fatigue, it is easy to cause the recurrence of pain symptoms in patients. Bone hyperplasia can be treated with oral non-steroidal anti-inflammatory and analgesic drugs and anti-bone hyperplasia medications. It is also necessary to reduce the amount of exercise, minimize local irritation, never get cold, and local heat application can be beneficial.
What should not be eaten with osteophyte formation?
Patients with bone hyperplasia should avoid spicy and stimulating foods, high-fat and high-calorie foods, such as fatty meat, barbecue, fried foods, and foods with high sugar content, as these are detrimental to health and can easily lead to diseases such as hypertension and hyperlipidemia. Patients with bone hyperplasia are encouraged to consume more calcium-rich foods, such as milk, shrimp shells, beans, and ribs. These foods are rich in calcium. Additionally, it is beneficial to get more sun exposure, adequately supplement with vitamin D, and engage in proper physical exercise, all of which help promote the absorption of calcium.