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Cerebral embolism
Clinical symptoms of cerebral embolism
Patients with cerebral embolism typically exhibit a sudden onset and rapid progression of the condition, often occurring during physical activity and rapidly reaching its peak. Clinically, this can result in immediate and complete paralysis of one side of the body or total aphasia, and in some cases, it can also lead to a swift onset of coma. Additionally, these patients often experience accompanying conditions such as arrhythmias, carotid artery plaque formation, and arteriosclerosis. The common sources of emboli in cerebral embolism are thrombi attached to the heart wall or plaques formed in the carotid artery. Therefore, these patients often have a history of hyperlipidemia, arteriosclerosis, plaque formation in arteries, atrial fibrillation, or coronary artery disease.
Auxiliary examinations for cerebral embolism
Patients with cerebral embolism need to undergo a cranial MRI to observe the location of the embolism, the area of brain tissue necrosis, and the age of the lesion, which is helpful in assessing the severity of the disease and prognosis. Additionally, it is necessary to examine the cervical vasculature with Doppler ultrasound to check for the presence of carotid artery plaques, especially soft plaques which are prone to detachment and can form emboli, blocking cerebral vessels and potentially causing recurrence or exacerbation of cerebral embolism. Furthermore, an echocardiogram of the heart is required because another common source of emboli in cerebral embolism is mural thrombi in the heart, particularly in patients with arrhythmias or atrial fibrillation, who are more prone to form mural thrombi. Therefore, patients with cerebral embolism need to have an echocardiogram to check for the presence of mural thrombi. If present, anticoagulant medication is required for treatment. (Please use medications under the guidance of a professional physician.)
How to treat cerebral embolism?
Brain embolism generally blocks relatively larger cerebral blood vessels and often leads to extensive death of brain cells. Therefore, patients with brain embolism are primarily treated with medications that promote blood circulation and remove blood stasis to improve circulation. Additionally, patients with brain embolism often experience an increase in intracranial pressure, so it is necessary to use medications that reduce intracranial pressure and alleviate brain cell edema. Patients with brain embolism might experience disturbances in consciousness and are prone to develop aspiration pneumonia or dependent pneumonia, so anti-infection medications may be utilized for treatment. Some may also suffer from stress-related gastrointestinal ulcers, or even gastrointestinal ulcer bleeding, thus requiring medications that inhibit gastric acid secretion and protect the gastric mucosa. Other treatments target the cause of the condition. A common cause is the detachment of carotid artery plaques, therefore, medications that prevent arteriosclerosis and stabilize plaques are also used. (Please use medications under the guidance of a doctor.)
Initial symptoms of cerebral embolism
The initial characteristics of cerebral embolism generally include a sudden onset and rapid progression of the condition. It often occurs during physical activity, with the condition reaching a peak in a very short time. Typically, within a short period, this can lead to complete paralysis of limbs on one side, complete loss of speech in some cases, and severe cases may quickly progress into a coma. The development of the condition is quite rapid. Common sources of emboli in cerebral embolism include mural thrombi from the heart and detached plaques from the carotid artery. Carotid artery plaques, especially soft plaques, are prone to detachment, forming emboli that block the cerebral vessels and cause cerebral embolism. Additionally, patients with arrhythmias or atrial fibrillation can easily form mural thrombi on the inner walls of the heart. Once a thrombus detaches, it can block cerebral vessels, leading to cerebral embolism. The affected area in cerebral embolism is relatively large, and the condition is quite severe.
What department should I go to for a cerebral embolism?
Brain embolism is a disease in the field of neurology, and one can consult a department of neurology for it. Patients with brain embolism often have a rapid onset, generally with a history of atrial fibrillation. Some thrombi attached to the heart dislodge into the cerebral arteries, causing brain embolism. Symptoms can rapidly develop, including limb paralysis and speech disorders. If the affected area in the brain is large, it may even lead to consciousness disorders and epileptic seizures. For the treatment of brain embolism, interventional thrombectomy can be performed, but there is a strict limitation on the timing window. It should be noted that patients with brain embolism have a high probability of transforming into cerebral hemorrhage, and caution must be exercised during treatment.
Can people with cerebral embolism run?
Patients with cerebral embolism can jog moderately, but walking is highly recommended. This is because the common source of the embolus in cerebral embolism patients is a mural thrombus in the heart. The common causes of mural thrombus formation include arrhythmia and atrial fibrillation. Therefore, patients with cerebral embolism, who may also have coronary artery disease, atrial fibrillation, or cardiac mural thrombus, are not well-suited for running, which is a relatively intense form of exercise. They can jog moderately, but the distance and duration should not be too long. The most recommended form of exercise is walking. Walking is the best form of exercise to prevent worsening the condition after running, and it could also exacerbate coronary artery disease conditions.
Can people with cerebral embolism eat lamb?
Patients with cerebral embolism can appropriately eat some mutton, but not too much, as the main dietary approach is a low-salt diet. Additionally, foods high in fat should not be consumed excessively, because a common cause of cerebral embolism is the hardening of blood vessels in the neck, carotid artery plaques, especially when soft plaques break off and form emboli that block cerebral vessels through the bloodstream. Thus, consuming these high-fat foods excessively can exacerbate hyperlipidemia, worsen arterial sclerosis, and increase arterial plaque, potentially leading to the recurrence and aggravation of cerebral embolism. Therefore, patients with cerebral embolism can appropriately consume some mutton, as its fat content is not too high, but it should not be consumed in excess. Eating it in moderation is acceptable.
Can cerebral embolism be treated with thrombolysis?
Cerebral embolism is a relatively common ischemic cerebrovascular disease. If the area of cerebral embolism is very large and it exceeds the time window for thrombolysis, and if the patient is elderly with multiple underlying diseases, then thrombolysis is not recommended. Patients with large-area cerebral embolism have a higher probability of transitioning to cerebral hemorrhage, thus the risk of thrombolysis is very high. However, if it is within the time window for arterial thrombectomy, it is advocated to undergo arterial thrombectomy treatment. Family members can communicate more with the doctor to choose the appropriate treatment plan. If the symptoms of cerebral embolism are relatively mild, the affected area is considered small, and it is within three hours, then intravenous thrombolysis treatment could be considered. Patients undergoing intravenous thrombolysis must ensure that a cranial CT is rechecked within 24 hours to exclude any secondary bleeding.
Symptoms and manifestations of cerebral embolism
The so-called cerebral embolism primarily refers to the presence of abnormal substances in the arteries, which block the smaller arteries leading to arterial occlusion, causing severe cerebral ischemia and hypoxia, and resulting in the necrosis of brain tissue. It generally occurs in patients with atrial fibrillation, where atrial fibrillation can form larger emboli that detach from the heart and can easily cause cerebral embolism. The symptoms of the onset are very sudden and rapidly reach their peak. Patients may experience coma and consciousness disorders, fixed staring of the eyes, paralysis of limbs, numbness of limbs, and an inability to sense pain and temperature in the limbs. Additionally, some may experience epileptic seizures and urinary and fecal incontinence. Cerebral embolism is a very dangerous disease, and once these symptoms appear, it is crucial to seek hospital treatment as soon as possible.
Causes of cerebral embolism
Any abnormal substance entering the cerebral arteries causing ischemia, hypoxia, and necrosis of brain tissue is called cerebral embolism. The causes of cerebral embolism mainly include the following types: The first type is cardiogenic cerebral embolism, which is caused by heart diseases. For example, the most common atrial fibrillation is the most common cause of cerebral embolism. Atrial fibrillation can lead to the formation of some mural thrombi. These thrombi form, detach, and enter the cerebral arteries causing embolism. Additionally, myocardial infarction is also an important cause, and patients with myocardial infarction can also form some abnormal thrombi in the heart. Second, other causes, such as air embolism. In patients with fractures, a large amount of fatty tissue enters the cerebral arteries, which can also cause cerebral arterial embolism. Moreover, some venous thrombi from the lower limbs can also form cerebral embolism. During childbirth, the entry of amniotic fluid into the brain can also cause cerebral embolism.