Initial symptoms of cerebral embolism

Written by Liu Yan Hao
Neurology
Updated on December 29, 2024
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The initial characteristics of cerebral embolism generally include a sudden onset and rapid progression of the condition. It often occurs during physical activity, with the condition reaching a peak in a very short time. Typically, within a short period, this can lead to complete paralysis of limbs on one side, complete loss of speech in some cases, and severe cases may quickly progress into a coma. The development of the condition is quite rapid.

Common sources of emboli in cerebral embolism include mural thrombi from the heart and detached plaques from the carotid artery. Carotid artery plaques, especially soft plaques, are prone to detachment, forming emboli that block the cerebral vessels and cause cerebral embolism. Additionally, patients with arrhythmias or atrial fibrillation can easily form mural thrombi on the inner walls of the heart. Once a thrombus detaches, it can block cerebral vessels, leading to cerebral embolism. The affected area in cerebral embolism is relatively large, and the condition is quite severe.

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Written by Zhang Hui
Neurology
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What to do about cerebral embolism?

The onset of cerebral embolism is very sudden, and most cases of cerebral embolism are caused by heart disease. For instance, atrial fibrillation or myocardial infarction can lead to cerebral embolism, which requires immediate medical attention. If it is within the time window for arterial thrombectomy, it is best to proceed with the arterial thrombectomy treatment, and this window typically ranges from six to eight hours. If the time window is missed, the patient should primarily be kept on bed rest and given medications to protect and nourish brain cells. If intracranial pressure is high, medications to reduce dehydration and lower cranial pressure should be administered. After the acute phase, anticoagulation treatment should also be provided to prevent future occurrences of cerebral embolism. Additionally, care should be taken to prevent complications such as pulmonary infections, urinary system infections, and bedsores.

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Written by Zhang Hui
Neurology
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Can people with cerebral embolism eat beef?

Patients with cerebral embolism can eat beef, as it contains rich high-quality proteins. These proteins can enhance the body's resistance and prevent complications such as lung and urinary tract infections following a cerebral embolism. Additionally, beef is rich in B vitamins such as vitamin B1 and B12, which also nourish the nerves. Cerebral embolism is an acute disease that often strikes suddenly, generally in patients with a history of atrial fibrillation, presenting with rapid onset and paralysis of limbs. Treatment involves timely administration of anticoagulants and statin drugs. Furthermore, after stabilization of the condition, active rehabilitation training is important. In terms of diet, in addition to beef, patients can also consume milk and eggs and should eat a variety of fresh vegetables and fruits. (Note: The answer is for reference only, please use medication under the guidance of a professional physician and do not self-medicate.)

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Written by Zhang Hui
Neurology
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Can people with cerebral embolism eat pork?

Patients with cerebral embolism generally suffer from heart diseases. For example, myocardial infarction can cause mural thrombi or atrial fibrillation, leading to thrombi in the left atrial appendage. These thrombi may detach and enter the cerebral arteries, resulting in cerebral embolism. The onset is quite severe, with patients rapidly developing symptoms that quickly peak. Clinical manifestations may include speech impairment, limb paralysis, and numbness. In severe cases, due to extensive damage, patients may experience coma or even death. Patients with cerebral embolism can consume small amounts of pork, but it is advisable to eat mainly lean pork. Lean pork contains some protein and B vitamins, which are beneficial for brain recovery. It is not advisable to eat fatty parts, as they can increase blood lipids and lead to arteriosclerosis.

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Written by Liu Yan Hao
Neurology
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Auxiliary examinations for cerebral embolism

Patients with cerebral embolism need to undergo a cranial MRI to observe the location of the embolism, the area of brain tissue necrosis, and the age of the lesion, which is helpful in assessing the severity of the disease and prognosis. Additionally, it is necessary to examine the cervical vasculature with Doppler ultrasound to check for the presence of carotid artery plaques, especially soft plaques which are prone to detachment and can form emboli, blocking cerebral vessels and potentially causing recurrence or exacerbation of cerebral embolism. Furthermore, an echocardiogram of the heart is required because another common source of emboli in cerebral embolism is mural thrombi in the heart, particularly in patients with arrhythmias or atrial fibrillation, who are more prone to form mural thrombi. Therefore, patients with cerebral embolism need to have an echocardiogram to check for the presence of mural thrombi. If present, anticoagulant medication is required for treatment. (Please use medications under the guidance of a professional physician.)

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Written by Tang Li Li
Neurology
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What department should one go to for a cerebral embolism?

Patients with cerebral embolism should visit the department of neurology. Cerebral embolism is not a special disease but rather one type of cerebral infarction. Cerebral infarction generally includes two types: cerebral thrombosis and cerebral embolism. The former refers to the formation of a thrombus at the site of the vessel occlusion. The latter involves a thrombus originating from another location, which blocks the vessel at the infarct site. The sources of such thrombi are varied, with the most common being from the heart, frequently seen in patients with long-term chronic atrial fibrillation. This condition forms a mural thrombus in the atrium, which, during episodes of atrial fibrillation, can detach, be flushed by the blood stream into the brain, and cause cerebral embolism. Secondly, it occurs in cardiac valve diseases, such as rheumatic heart disease, mitral valve alterations, and others. There are also some other sources of thrombi, such as tumor-induced cancer, amniotic fluid embolism in pregnant women, and fat embolism in patients with fractures.