Can people with cerebral embolism eat beef?

Written by Zhang Hui
Neurology
Updated on September 05, 2024
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Patients with cerebral embolism can eat beef, as it contains rich high-quality proteins. These proteins can enhance the body's resistance and prevent complications such as lung and urinary tract infections following a cerebral embolism. Additionally, beef is rich in B vitamins such as vitamin B1 and B12, which also nourish the nerves. Cerebral embolism is an acute disease that often strikes suddenly, generally in patients with a history of atrial fibrillation, presenting with rapid onset and paralysis of limbs. Treatment involves timely administration of anticoagulants and statin drugs. Furthermore, after stabilization of the condition, active rehabilitation training is important. In terms of diet, in addition to beef, patients can also consume milk and eggs and should eat a variety of fresh vegetables and fruits. (Note: The answer is for reference only, please use medication under the guidance of a professional physician and do not self-medicate.)

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Written by Zhang Hui
Neurology
1min 3sec home-news-image

Is cerebral embolism the same as cerebral infarction?

Brain embolism is a form of cerebral infarction, so essentially, it is also a type of cerebral infarction. Cerebral infarction encompasses a wider range, including cerebral thrombosis, lacunar infarction, watershed infarction, and others. Brain embolism primarily refers to abnormal substances entering the bloodstream, which then enter the arteries of the brain, causing obstruction in these arteries and leading to ischemia and hypoxia of the brain tissue, thus presenting clinical symptoms of cerebral infarction. The onset of the condition in patients is quite severe, rapidly leading to paralysis of limbs and disorders of speech function. In cases of extensive brain embolism, patients may even experience coma and death as serious complications. Most patients with brain embolism have a history of atrial fibrillation. Atrial fibrillation can lead to the formation of mural thrombi, and when these thrombi dislodge, they can cause brain embolism.

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Written by Zhang Hui
Neurology
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How many days for brain embolism infusion?

Cerebral embolism is a particularly dangerous disease that is very concentrated. It primarily refers to the entry of abnormal substances into the arteries, leading to the occlusion of the cerebral arteries and causing corresponding clinical manifestations in patients. Symptoms may include disturbances in consciousness, coma, limb paralysis, numbness, slurred speech, and even possibly seizures. The most common cause of cerebral embolism is atrial fibrillation. Treatment generally requires hospitalization for intravenous infusion. If the condition is relatively stable, an infusion lasting about 10 to 14 days may suffice, followed by administration of anticoagulant drugs for treatment. If the area affected by the cerebral embolism is very large and the patient is in severe danger, infusion treatment may need to last about 3 to 4 weeks. The specific approach should be based on the patient's condition. Additionally, certain medications to reduce dehydration and intracranial pressure, as well as neuroprotective drugs, should be administered. (Please use medication under the guidance of a doctor.)

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Written by Zhang Hui
Neurology
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Which department should I go to for a cerebral embolism?

For most hospitals, registration for cerebral embolism requires a visit to the Department of Neurology, which is the most specialized department for treating cerebral embolism. Cerebral embolism generally has corresponding causes, with the most common being heart diseases. For example, conditions such as old myocardial infarction or atrial fibrillation can easily form mural thrombi. These thrombi can detach, enter the cranial arteries through circulation, and cause cerebral embolism, leading to severe clinical consequences. The onset of the disease is very aggressive, quickly reaching a peak and resulting in limb paralysis or even consciousness disorders, requiring emergency treatment. If it is within the thrombolytic time window, intravenous thrombolytic therapy can be administered after assessment. However, one must be cautious of the risk of hemorrhagic transformation during thrombolysis for cerebral embolism. If within a certain timeframe, arterial thrombectomy can also be performed, and many in the Department of Neurology are capable of conducting such surgeries.

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Written by Liu Yan Hao
Neurology
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Auxiliary examinations for cerebral embolism

Patients with cerebral embolism need to undergo a cranial MRI to observe the location of the embolism, the area of brain tissue necrosis, and the age of the lesion, which is helpful in assessing the severity of the disease and prognosis. Additionally, it is necessary to examine the cervical vasculature with Doppler ultrasound to check for the presence of carotid artery plaques, especially soft plaques which are prone to detachment and can form emboli, blocking cerebral vessels and potentially causing recurrence or exacerbation of cerebral embolism. Furthermore, an echocardiogram of the heart is required because another common source of emboli in cerebral embolism is mural thrombi in the heart, particularly in patients with arrhythmias or atrial fibrillation, who are more prone to form mural thrombi. Therefore, patients with cerebral embolism need to have an echocardiogram to check for the presence of mural thrombi. If present, anticoagulant medication is required for treatment. (Please use medications under the guidance of a professional physician.)

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Written by Tang Li Li
Neurology
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What department should one go to for a cerebral embolism?

Patients with cerebral embolism should visit the department of neurology. Cerebral embolism is not a special disease but rather one type of cerebral infarction. Cerebral infarction generally includes two types: cerebral thrombosis and cerebral embolism. The former refers to the formation of a thrombus at the site of the vessel occlusion. The latter involves a thrombus originating from another location, which blocks the vessel at the infarct site. The sources of such thrombi are varied, with the most common being from the heart, frequently seen in patients with long-term chronic atrial fibrillation. This condition forms a mural thrombus in the atrium, which, during episodes of atrial fibrillation, can detach, be flushed by the blood stream into the brain, and cause cerebral embolism. Secondly, it occurs in cardiac valve diseases, such as rheumatic heart disease, mitral valve alterations, and others. There are also some other sources of thrombi, such as tumor-induced cancer, amniotic fluid embolism in pregnant women, and fat embolism in patients with fractures.