Which department should I go to for a cerebral embolism?

Written by Zhang Hui
Neurology
Updated on September 07, 2024
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For most hospitals, registration for cerebral embolism requires a visit to the Department of Neurology, which is the most specialized department for treating cerebral embolism. Cerebral embolism generally has corresponding causes, with the most common being heart diseases. For example, conditions such as old myocardial infarction or atrial fibrillation can easily form mural thrombi. These thrombi can detach, enter the cranial arteries through circulation, and cause cerebral embolism, leading to severe clinical consequences. The onset of the disease is very aggressive, quickly reaching a peak and resulting in limb paralysis or even consciousness disorders, requiring emergency treatment. If it is within the thrombolytic time window, intravenous thrombolytic therapy can be administered after assessment. However, one must be cautious of the risk of hemorrhagic transformation during thrombolysis for cerebral embolism. If within a certain timeframe, arterial thrombectomy can also be performed, and many in the Department of Neurology are capable of conducting such surgeries.

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Written by Liu Yan Hao
Neurology
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Auxiliary examinations for cerebral embolism

Patients with cerebral embolism need to undergo a cranial MRI to observe the location of the embolism, the area of brain tissue necrosis, and the age of the lesion, which is helpful in assessing the severity of the disease and prognosis. Additionally, it is necessary to examine the cervical vasculature with Doppler ultrasound to check for the presence of carotid artery plaques, especially soft plaques which are prone to detachment and can form emboli, blocking cerebral vessels and potentially causing recurrence or exacerbation of cerebral embolism. Furthermore, an echocardiogram of the heart is required because another common source of emboli in cerebral embolism is mural thrombi in the heart, particularly in patients with arrhythmias or atrial fibrillation, who are more prone to form mural thrombi. Therefore, patients with cerebral embolism need to have an echocardiogram to check for the presence of mural thrombi. If present, anticoagulant medication is required for treatment. (Please use medications under the guidance of a professional physician.)

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Written by Liu Yan Hao
Neurology
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Initial symptoms of cerebral embolism

The initial characteristics of cerebral embolism generally include a sudden onset and rapid progression of the condition. It often occurs during physical activity, with the condition reaching a peak in a very short time. Typically, within a short period, this can lead to complete paralysis of limbs on one side, complete loss of speech in some cases, and severe cases may quickly progress into a coma. The development of the condition is quite rapid. Common sources of emboli in cerebral embolism include mural thrombi from the heart and detached plaques from the carotid artery. Carotid artery plaques, especially soft plaques, are prone to detachment, forming emboli that block the cerebral vessels and cause cerebral embolism. Additionally, patients with arrhythmias or atrial fibrillation can easily form mural thrombi on the inner walls of the heart. Once a thrombus detaches, it can block cerebral vessels, leading to cerebral embolism. The affected area in cerebral embolism is relatively large, and the condition is quite severe.

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Written by Tang Bo
Neurology
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What should not be eaten in case of cerebral embolism?

Cerebral embolism is a type of cerebral infarction. It is a condition caused by the dislodgment of emboli, leading to the interruption of blood flow in the brain and resulting in the necrosis of brain tissue and cerebral infarction. There are no special dietary requirements for this condition, but it is generally advisable to follow a low-salt, low-fat diet, as high levels of salt or fats can be risk factors for cerebral infarction. Additionally, cerebral embolism can also be caused by atrial fibrillation. Patients with atrial fibrillation must monitor their coagulation function carefully and pay attention to cardiac issues. Regarding the diet, it should be low in salt and fat, and if the patient does not have diabetes, there is no need to pay special attention to blood sugar levels. However, if diabetes is present, diabetic dietary guidelines must be followed.

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Written by Liu Yan Hao
Neurology
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Clinical symptoms of cerebral embolism

Patients with cerebral embolism typically exhibit a sudden onset and rapid progression of the condition, often occurring during physical activity and rapidly reaching its peak. Clinically, this can result in immediate and complete paralysis of one side of the body or total aphasia, and in some cases, it can also lead to a swift onset of coma. Additionally, these patients often experience accompanying conditions such as arrhythmias, carotid artery plaque formation, and arteriosclerosis. The common sources of emboli in cerebral embolism are thrombi attached to the heart wall or plaques formed in the carotid artery. Therefore, these patients often have a history of hyperlipidemia, arteriosclerosis, plaque formation in arteries, atrial fibrillation, or coronary artery disease.

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Written by Zhang Hui
Neurology
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How many days for brain embolism infusion?

Cerebral embolism is a particularly dangerous disease that is very concentrated. It primarily refers to the entry of abnormal substances into the arteries, leading to the occlusion of the cerebral arteries and causing corresponding clinical manifestations in patients. Symptoms may include disturbances in consciousness, coma, limb paralysis, numbness, slurred speech, and even possibly seizures. The most common cause of cerebral embolism is atrial fibrillation. Treatment generally requires hospitalization for intravenous infusion. If the condition is relatively stable, an infusion lasting about 10 to 14 days may suffice, followed by administration of anticoagulant drugs for treatment. If the area affected by the cerebral embolism is very large and the patient is in severe danger, infusion treatment may need to last about 3 to 4 weeks. The specific approach should be based on the patient's condition. Additionally, certain medications to reduce dehydration and intracranial pressure, as well as neuroprotective drugs, should be administered. (Please use medication under the guidance of a doctor.)