How to treat cerebral embolism?

Written by Liu Yan Hao
Neurology
Updated on February 07, 2025
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Brain embolism generally blocks relatively larger cerebral blood vessels and often leads to extensive death of brain cells. Therefore, patients with brain embolism are primarily treated with medications that promote blood circulation and remove blood stasis to improve circulation. Additionally, patients with brain embolism often experience an increase in intracranial pressure, so it is necessary to use medications that reduce intracranial pressure and alleviate brain cell edema. Patients with brain embolism might experience disturbances in consciousness and are prone to develop aspiration pneumonia or dependent pneumonia, so anti-infection medications may be utilized for treatment. Some may also suffer from stress-related gastrointestinal ulcers, or even gastrointestinal ulcer bleeding, thus requiring medications that inhibit gastric acid secretion and protect the gastric mucosa. Other treatments target the cause of the condition. A common cause is the detachment of carotid artery plaques, therefore, medications that prevent arteriosclerosis and stabilize plaques are also used. (Please use medications under the guidance of a doctor.)

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Written by Zhang Hui
Neurology
50sec home-news-image

What department should I go to for a cerebral embolism?

Brain embolism is a disease in the field of neurology, and one can consult a department of neurology for it. Patients with brain embolism often have a rapid onset, generally with a history of atrial fibrillation. Some thrombi attached to the heart dislodge into the cerebral arteries, causing brain embolism. Symptoms can rapidly develop, including limb paralysis and speech disorders. If the affected area in the brain is large, it may even lead to consciousness disorders and epileptic seizures. For the treatment of brain embolism, interventional thrombectomy can be performed, but there is a strict limitation on the timing window. It should be noted that patients with brain embolism have a high probability of transforming into cerebral hemorrhage, and caution must be exercised during treatment.

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Written by Tang Li Li
Neurology
1min 17sec home-news-image

What department should one go to for a cerebral embolism?

Patients with cerebral embolism should visit the department of neurology. Cerebral embolism is not a special disease but rather one type of cerebral infarction. Cerebral infarction generally includes two types: cerebral thrombosis and cerebral embolism. The former refers to the formation of a thrombus at the site of the vessel occlusion. The latter involves a thrombus originating from another location, which blocks the vessel at the infarct site. The sources of such thrombi are varied, with the most common being from the heart, frequently seen in patients with long-term chronic atrial fibrillation. This condition forms a mural thrombus in the atrium, which, during episodes of atrial fibrillation, can detach, be flushed by the blood stream into the brain, and cause cerebral embolism. Secondly, it occurs in cardiac valve diseases, such as rheumatic heart disease, mitral valve alterations, and others. There are also some other sources of thrombi, such as tumor-induced cancer, amniotic fluid embolism in pregnant women, and fat embolism in patients with fractures.

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Written by Zhang Hui
Neurology
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What to do about cerebral embolism?

The onset of cerebral embolism is very sudden, and most cases of cerebral embolism are caused by heart disease. For instance, atrial fibrillation or myocardial infarction can lead to cerebral embolism, which requires immediate medical attention. If it is within the time window for arterial thrombectomy, it is best to proceed with the arterial thrombectomy treatment, and this window typically ranges from six to eight hours. If the time window is missed, the patient should primarily be kept on bed rest and given medications to protect and nourish brain cells. If intracranial pressure is high, medications to reduce dehydration and lower cranial pressure should be administered. After the acute phase, anticoagulation treatment should also be provided to prevent future occurrences of cerebral embolism. Additionally, care should be taken to prevent complications such as pulmonary infections, urinary system infections, and bedsores.

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Written by Liu Yan Hao
Neurology
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Initial symptoms of cerebral embolism

The initial characteristics of cerebral embolism generally include a sudden onset and rapid progression of the condition. It often occurs during physical activity, with the condition reaching a peak in a very short time. Typically, within a short period, this can lead to complete paralysis of limbs on one side, complete loss of speech in some cases, and severe cases may quickly progress into a coma. The development of the condition is quite rapid. Common sources of emboli in cerebral embolism include mural thrombi from the heart and detached plaques from the carotid artery. Carotid artery plaques, especially soft plaques, are prone to detachment, forming emboli that block the cerebral vessels and cause cerebral embolism. Additionally, patients with arrhythmias or atrial fibrillation can easily form mural thrombi on the inner walls of the heart. Once a thrombus detaches, it can block cerebral vessels, leading to cerebral embolism. The affected area in cerebral embolism is relatively large, and the condition is quite severe.

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Written by Zhang Hui
Neurology
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Can you exercise with a cerebral embolism?

Brain embolism is a form of cerebral infarction. The mechanism of its occurrence mainly involves abnormal objects entering the arteries, causing occlusion in the cerebral arteries and leading to brain embolism. Patients may experience paralysis, limb numbness, speech impairments, and in severe cases, even coma. The most common cause of brain embolism is due to atrial fibrillation or myocardial infarction causing mural thrombi. These dislodged thrombi travel to cerebral arteries, causing brain embolism. Generally, during the acute phase of brain embolism, bed rest is recommended along with some bedside functional exercises, such as active and passive movements of the limbs. Additionally, cardiac function must be considered since cardiac function is generally compromised in patients with brain embolism. If the treatment of brain embolism progresses well into the recovery or residual phase, and if cardiac function permits, moderate exercise such as brisk walking and slow jogging may be appropriate, but it is essential to protect the patient's cardiac function.