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Xie Zhi Hong
Cardiology
About me
The Secretary-General and Standing Committee Member of the Cardiovascular Disease Professional Committee of the Rehabilitation Medical Association in Ganzhou City, and a member of the Ganzhou City Working Committee of the Heart Emergency Branch of the China Medical Health International Exchange Promotion Association. He has chaired 4 provincial and municipal-level research projects, participated in more than 20 research projects, and published over 20 papers.
Proficient in diseases
Specializes in the diagnosis and treatment of coronary heart disease, hypertension, and structural heart disease. Research interests include hypertension, coronary heart disease, arrhythmia, and cardiac rehabilitation.
Voices
Will patent ductus arteriosus show signs of peripheral vascular disease?
Peripheral vascular signs refer to a medical sign characterized by enhanced pulsation of the peripheral arteries and capillaries due to increased pulse pressure. Patients with a patent ductus arteriosus can experience increased pulse pressure, leading to rapid rises and falls in pressure within the peripheral vessels, thereby causing a series of peripheral vascular signs. When a patent ductus arteriosus is combined with peripheral vascular signs, indicating a substantial shunt, it is advisable for such individuals to undergo surgery as soon as possible. Delaying surgery may lead to a right-to-left shunt, resulting in Eisenmenger's syndrome, and the loss of the opportunity for surgical intervention.
Is congenital heart disease with perimembranous ventricular septal defect serious?
Simple congenital heart defects, such as perimembranous defects, generally are not particularly severe. However, there are several scenarios to consider: The first scenario involves a large defect that typical occluders cannot address; in such cases, surgical intervention is required. This type of defect is generally considered severe. Another scenario is when a cardiac defect has been present for an extended period, leading to repeated shortness of breath, heart failure, cardiac enlargement, and even pulmonary arterial hypertension. This situation would be considered a severe ventricular septal defect, caused by the failure to treat the membranous part of the septal defect in time. Thus, the majority of ventricular septal defects are not very severe in the early stages. They become serious if the defect is too large or if there is coexistence with other defects, such as an overriding aorta or poorly developed pulmonary artery, among other complex congenital heart diseases. Regardless, patients with perimembranous congenital heart defects should undergo surgical treatment early to avoid missing the opportunity for surgery.
Causes of recurrent pericardial effusion
Pericardial effusion is generally seen in inflammatory responses, such as nonspecific bacterial infections, as well as tuberculosis, chlamydia, mycoplasma, rheumatic heart disease, and it can also be caused by heart failure, uremia leading to pericardial effusion. Some are caused by tumors. In general, common nonspecific infections and tuberculosis can be quickly controlled with treatment, but in cases of tumors or due to various reasons such as hypoalbuminemia, heart failure, if the cause cannot be eliminated, pericardial effusion can frequently reoccur. Therefore, individuals with recurring pericardial effusion should go to the hospital for an examination to clarify the cause and rule out malignant tumors or other reasons, and receive timely treatment.
Can patients with rheumatic heart disease not drink a lot of water?
The early manifestations of rheumatic heart disease include rheumatic myocarditis or pericarditis, sometimes accompanied by significant pericardial effusion. In later stages, there may be thickening of the pericardium, presenting as constrictive pericarditis. Some patients may also develop severe mitral stenosis, aortic stenosis, or insufficiency. When the disease reaches a certain level of severity, the patient can experience cardiac failure and systemic sodium and water retention. In such cases, it becomes necessary to control water intake. Therefore, in the early stages of rheumatic heart disease when there is no cardiac failure, it is possible to drink more water. However, when patients experience severe cardiac failure, edema, and similar conditions, it is not advisable to drink excessive amounts of water.
Is myocarditis cough severe?
Patients with myocarditis often exhibit early symptoms such as fever, cough, difficulty breathing, fatigue, and in severe cases, chest tightness and shortness of breath, even leading to shock or death, and syncope. Typically, the cough is not too severe, but if myocarditis is suspected, the patient should go to the hospital for timely diagnosis. Primary diagnostic tests include electrocardiogram, myocardial enzymes, and cardiac ultrasound. Once diagnosed, hospitalization for immediate treatment is necessary to prevent the condition from worsening. Because fulminant myocarditis can lead to death within days from minor symptoms, all myocarditis patients should be treated as severe cases and not be neglected.
How long does the pain from a myocardial infarction last?
Acute myocardial infarction often develops from angina pectoris. When angina persists for more than 30 minutes, myocardial necrosis begins. After the myocardial necrosis, symptoms similar to those of a burn appear, such as reddening of the myocardium, blistering, and necrosis. Generally speaking, these symptoms last no less than three days. Many people experience significant improvement after three days because the area of myocardial infarction is relatively small. In patients with extensive myocardial infarction, symptoms can last for 7 days or even longer. During this time, patients may also experience respiratory difficulties, recurrent chest pain, and even sudden death. Therefore, the duration of symptoms in myocardial infarction generally ranges from 3 to 7 days. If there is no improvement within this period, the disease might have led to severe complications.
Can heart failure cause suffocation?
Heart failure is caused by various reasons leading to the heart's blood supply being relatively or absolutely insufficient, causing a series of pathological and physiological disorders in patients. Patients often manifest symptoms like difficulty breathing, tightness in breath, and even a feeling of suffocation. However, generally, the symptoms caused by heart failure are not these. This is because suffocation occurs due to airway narrowing or the narrow space around the patient, leading to insufficient oxygen intake, causing a series of syndromes. Therefore, although people with heart failure feel suffocated, they are not actually suffocating; it is just a manifestation of inadequate circulatory supply.
Mitral stenosis which ventricle hypertrophy
Patients with mitral stenosis need to pass the blood from the atrium to the heart through the mitral valve during cardiac diastole. The narrowing condition in patients with mitral stenosis leads to an accelerated and prolonged flow of blood from the atrium to the ventricle. As a result, atrial pressure increases, and this heightened atrial pressure can cause pulmonary congestion. In severe cases, it leads to pulmonary hypertension. Pulmonary hypertension, in turn, can cause enlargement of the right ventricle and tricuspid regurgitation, which can lead to the patient exhibiting cyanosis. Therefore, in patients with mitral stenosis, the atrium gradually enlarges in the early stages, and later stages may present with right ventricular hypertrophy.
Is vomiting severe in myocardial infarction?
Patients with myocardial infarction often have a history of hypertension, coronary heart disease, and diabetes. They usually experience repeated episodes of angina. After angina, the chest pain persists and cannot be alleviated, often including feelings of chest oppression and pressure. Some patients may also experience nausea, vomiting, and fainting, but most do not have severe vomiting. Some patients, because of inferior wall myocardial infarction or posterior wall myocardial infarction, may develop right ventricular dysfunction, or during the treatment process, due to decreased appetite, may suffer from severe vomiting caused by low potassium and low sodium levels. However, this can generally be corrected through treatment.
Can you drink alcohol with a patent ductus arteriosus?
Under normal circumstances, the ductus arteriosus in newborns gradually closes, and in most infants, the ductus arteriosus is completely closed within three months. However, for some people, even after one year of birth, the ductus arteriosus may not necessarily be diagnosed as congenital patent ductus arteriosus (PDA), a heart condition. Patients with this condition often experience pathological cardiac phenomena such as left-to-right shunts or right-to-left shunts. Due to these potential issues, the cardiac workload can worsen, leading to cardiac dysfunction and eventually heart failure. Drinking alcohol can potentially increase the patient's blood pressure and heart rate, adding more strain to the heart and exacerbating the condition. Therefore, it is advised that such patients avoid alcohol until after surgical treatment has been completed and their condition has stabilized. There are two methods of surgical treatment: most cases can be completely cured with interventional therapy, while a minority may require surgical ligation.