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Zhang Hui
Neurology
About me
Weifang People's Hospital, Department of Neurology, attending physician, has been engaged in clinical work in the field of neurology for many years, with rich clinical experience in common and prevalent neurological diseases.
Proficient in diseases
Cerebrovascular disease, Parkinson's disease, myelitis, etc.
Voices
Early symptoms of Parkinson's disease.
Parkinson's disease is a relatively common degenerative disease in the elderly, primarily causing the death of dopamine neurons in the substantia nigra of the midbrain, leading to corresponding clinical manifestations. Research has confirmed that other systems may also be damaged in the early stages of Parkinson's disease symptoms. The early symptoms may primarily include: First, patients experience persistent constipation, which is a very important early symptom of Parkinson's disease. Second, some patients may experience sleep disturbances, mainly characterized by tossing and turning, shouting out loud during sleep, without being aware of it. Additionally, some patients in the early stages may also experience a reduced sense of smell, constipation, cognitive impairment, and other clinical manifestations. There are also some early motor symptoms, mainly that the patient becomes clumsy and inflexible in the movements of one side of the body. These are the early symptoms of Parkinson's disease.
Is senile dementia hereditary?
Dementia primarily refers to Alzheimer's disease, which mainly has two types: The first type is sporadic Alzheimer's disease, which does not have a clear familial history. It is caused by the degeneration and death of neurons due to various reasons, leading to certain clinical symptoms. This type of sporadic dementia does not have a genetic predisposition. The other type of dementia has a clear family history, where patients typically exhibit mutations in the APP gene, leading to increased secretion, which in turn causes the death of neurons and results in the symptoms of dementia. This familial type of dementia has a genetic predisposition. Dementia is a common condition in neurology and is the most common neurodegenerative disease. Currently, there are no effective treatments available. Family members must pay attention to caregiving, and patients can take cholinesterase inhibitors, which can improve symptoms to a certain extent.
What department should Parkinson's disease go to?
Parkinson's disease is classified as a neurological disorder, so once considered for Parkinson's disease, registration is naturally in the department of neurology. Many neurologists are very familiar with Parkinson's disease and also have rich experience in diagnosis and treatment. Especially some neurology experts who specialize in Parkinson's disease have their own unique views on the pathogenesis, etiology, and clinical manifestations of the disease. Parkinson's disease is a chronic, degenerative neurological condition primarily divided into motor symptoms and non-motor symptoms, severely impacting the quality of life and work of patients. Symptoms include bradykinesia, resting tremor, muscle rigidity, etc. Treatment involves anticholinergic drugs and dopamine receptor agonists. (Note: This answer is for reference only. Medication should be administered under the guidance of a professional physician, and self-medication should be avoided.)
Symptoms of dementia in the elderly
Dementia in the elderly is medically known as Alzheimer's disease, which is categorized into mild, moderate, and severe stages. The clinical manifestations vary with the severity of the disease. Mild dementia primarily presents as memory impairment, starting with a decline in short-term memory, where patients often forget daily activities. For instance, they might forget the way home when they go out, forget to add salt when cooking, or forget to turn off the stove. Patients may also exhibit personality disorders, such as neglecting personal hygiene or not wanting to bathe. As the disease progresses to moderate dementia, besides worsening memory impairment, the patient's ability to work, learn new knowledge, and communicate with others significantly diminishes. There is a notable decline in previously acquired knowledge and skills, such as judgment and calculation abilities. They may also experience aphasia, apraxia, agnosia, and some patients may exhibit significant behavioral and psychological abnormalities. With further progression to severe dementia, patients may become emotionally detached, exhibit unpredictable crying or laughing, fail to recognize family members, lose verbal abilities, and even become unable to perform simple tasks such as dressing or eating. They may spend their days in bed, not speaking, and gradually lose contact with the outside world, ultimately leading to death.
Myasthenia Gravis Test Items
Myasthenia gravis is a common neurological disease, primarily an autoimmune disorder. It typically results in symptoms that are worse in the evening and fluctuate throughout the day, mainly manifesting as limb weakness, double vision, swallowing difficulties, and speech impairments. In severe cases, it can even affect respiration, leading to weakness of the respiratory muscles and necessitating the assistance of a ventilator. The key examinations for myasthenia gravis include: Firstly, a chest CT scan. Many patients with myasthenia gravis have thymic hyperplasia or thymomas, making this scan crucial. Secondly, repetitive nerve stimulation electromyography. This test can detect amplitude decrement in response to stimulation, which is significant for diagnosing the disease. Thirdly, testing for specific antibodies related to myasthenia gravis, such as acetylcholine receptor antibodies. The presence of these antibodies is very important for diagnosis. Additionally, other tests such as erythrocyte sedimentation rate and thyroid function tests are also conducted.
Can migraines cause vomiting?
Patients with migraines may experience vomiting, which is one of the most common complications of migraines. The occurrence of vomiting in migraines is mainly because the headache may affect the vomiting center in the brainstem, such as affecting the solitary nucleus. Damage to the solitary nucleus can lead to gastrointestinal dysfunction and result in vomiting. Many patients find that their headache symptoms are alleviated after vomiting. In terms of treatment, on one hand, it is necessary to correct the occurrence of migraines, mainly by administering pain relief medications and allowing the patient to rest in a quiet environment. For vomiting, antiemetic medications can be given to the patient, either orally or by muscle injection. Most patients show significant clinical improvement after treatment. (Please use medication under the guidance of a doctor.)
Early symptoms of myasthenia gravis
Myasthenia gravis is an autoimmune disease of the nervous system that primarily affects the acetylcholine receptors on the postsynaptic membrane, causing muscle fatigue in patients. This can lead to general weakness throughout the body, double vision, and even progressive paralysis of the respiratory muscles, requiring active breathing assistance in severe cases. The early symptoms of myasthenia gravis mainly include: First, patients tend to fatigue easily, such as after exercise or labor, with a rapid decline in physical strength that only recovers after resting. Second, there may be ptosis, with patients unable to comfortably open their eyes, although this symptom can improve after resting or sleeping. As myasthenia gravis progresses into its later stages, there is a marked increase in overall weakness, necessitating treatment in a neurology department.
What are the symptoms of cerebral atrophy?
Some patients with brain atrophy may not exhibit any specific clinical symptoms and are only found to have brain atrophy through imaging studies, such as magnetic resonance imaging (MRI) of the brain. Some patients do exhibit clinical symptoms, and the common symptoms of brain atrophy mainly include the following manifestations. First, patients may experience a decline in memory function, such as frequently forgetting things they have just done, forgetting where they placed their wallet, or forgetting to bring keys when going out. Second, there may also be a decline in executive functions, such as difficulties in dressing or brushing teeth by oneself. Third, there may be a decline in computational abilities, to the extent that the patient cannot perform simple arithmetic. Additionally, brain atrophy may also present some psychiatric symptoms, such as hallucinations and incoherent speech.
What vitamins to take for optic neuritis
Optic neuritis is primarily caused by the demyelination of the optic nerve. It is a type of central nervous system demyelinating disease. Therefore, patients with optic neuritis can take some vitamins, mainly B vitamins, including vitamin B12, vitamin B6, vitamin B1, etc. These can nourish the nerves and promote the regeneration of the myelin sheath. In addition, patients with optic neuritis also need to use some corticosteroids. Corticosteroids mainly suppress the immune response and inhibit the body's antibodies from damaging the myelin sheath of the optic nerve, playing a very important role in foundational treatment. Additionally, some severe cases of optic neuritis may even require the use of immunoglobulin and other immunosuppressive agents are also used in patients with optic neuritis.
Can optic neuritis recover by itself?
Optic neuritis is a demyelinating disease of the central nervous system, caused by an immune-inflammatory response of the body. Most patients with optic neuritis have severe immune dysfunction, making full recovery on their own very difficult. Although a very small number are fortunate enough to potentially self-heal without specific treatment, such cases are rare. Treatment for optic neuritis is essential because if left untreated, it could likely lead to permanent vision reduction. Additionally, the disease may also affect the brain and spinal cord, leading to clinical manifestations such as paralysis of limbs. Therefore, it is advisable to actively treat this condition. The primary treatment involves administration of corticosteroids and possibly long-term oral intake of immunomodulators to prevent recurrence. (Medications should be used under the guidance of a doctor based on specific conditions.)