The causes of nausea and vomiting in acute pancreatitis

Written by He Zong Quan
General Surgery
Updated on October 22, 2024
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The causes of acute pancreatitis are often considered to be gallstones, as well as hyperlipidemia, alcohol-related reasons, and others. After the onset of pancreatitis, it usually causes significant gastrointestinal symptoms, accompanied by nausea and vomiting. Due to the increased intra-abdominal pressure, it can also cause abdominal compartment syndrome, which can exacerbate gastrointestinal symptoms. Therefore, the symptoms of nausea and vomiting may worsen, and after vomiting, symptoms do not ease significantly, which is a clear distinction from other diseases. After the onset of acute pancreatitis, it is necessary to address the root cause of the condition, and to use acid suppression, enzyme inhibition, and suppression of gastrointestinal symptoms to promote the recovery from pancreatitis.

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Does acute pancreatitis cause abdominal muscle tension?

If a patient with acute pancreatitis shows symptoms of peritonitis, abdominal muscle rigidity may occur. Acute pancreatitis is divided into acute edematous pancreatitis and acute hemorrhagic necrotizing pancreatitis. The main symptoms of acute edematous pancreatitis include abdominal pain, nausea, vomiting, and fever. If it is hemorrhagic necrotizing pancreatitis, it can lead to shock, respiratory distress syndrome, high fever, jaundice, abdominal distension, and the abdominal compartment syndrome in the patient. Severe cases can cause intestinal paralysis and signs of peritoneal irritation, leading to abdominal muscle rigidity and also possibly presenting with shifting dullness, among other symptoms.

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Is acute severe pancreatitis serious?

Acute severe pancreatitis is a very serious disease in clinical practice, with a high mortality rate, often requiring comprehensive treatment in the ICU. Acute severe pancreatitis is a disease caused by multiple etiologies that results in localized inflammation, necrosis, and infection of the pancreas, accompanied by a systemic inflammatory response and persistent organ failure. It is divided into three phases. The first phase is the acute response phase, occurring from onset to about two weeks, characterized by systemic inflammatory response; the second phase is the systemic infection phase, occurring from two weeks to about two months, characterized by necrosis and infection of the pancreas or peripancreatic tissues; the third phase is the residual infection phase, occurring two to three months later, with clinical manifestations primarily of systemic malnutrition and persistent fistulas, accompanied by gastrointestinal fistulas.

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What department should I go to for acute pancreatitis?

Acute pancreatitis can be treated at the Department of Gastroenterology in local hospitals. It commonly begins with abdominal pain clinically, so it is recommended that patients first visit the Department of Gastroenterology. The pain from acute pancreatitis is primarily in the upper abdomen, persistent, and may radiate to the back. Of course, some patients may experience nausea, vomiting, cessation of gas and bowel movements, fever, etc. For such patients, it is recommended to complete examinations such as blood amylase, urine amylase, and lipase. When necessary, an abdominal ultrasound and CT scan should be conducted to assess the condition of the pancreatitis. Diagnosing pancreatitis is generally straightforward. As for treatment, it mainly involves internal medicine pharmacotherapy, fasting, fluid replacement, gastrointestinal decompression, and suppressing pancreatic secretion, among others. Of course, if complications arise, surgical intervention by the Department of Hepatobiliary Surgery may be required. (The use of medication should be under the guidance of a doctor.)

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What are the clinical manifestations of acute pancreatitis?

Acute pancreatitis includes two main types: acute edematous pancreatitis and acute necrotizing pancreatitis. It is a common acute and critical condition of the digestive system, particularly acute necrotizing pancreatitis, which can often be life-threatening. The primary symptoms usually include severe upper left abdominal pain, fever, chills, shivering, vomiting, etc. There is a significant relationship between abdominal pain and eating; generally, the pain worsens after eating and can sometimes radiate to the back. The main causes could be binge drinking, overeating, cholecystitis, gallstones, or hyperlipidemia, among others. Diagnosis is usually confirmed through examinations such as an upper abdominal CT and serum and urine amylase tests, followed by appropriate treatment measures.

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How long after being discharged with acute pancreatitis can one resume a normal diet?

After being discharged from the hospital, patients with acute pancreatitis, upon re-examination of routine blood tests and upper abdominal CT scans showing complete healing of pancreatitis, may, as appropriate, consume a low-fat, high-carbohydrate diet and gradually transition back to a normal diet. This process generally requires at least two months to be considered safe. Patients discharged after acute pancreatitis must abstain strictly from alcohol, avoid binge eating, and avoid overly greasy food. Greasy food can easily lead to increased bile discharge and potentially cause blockage of the pancreas, possibly triggering a recurrence of pancreatitis.