Neonatal jaundice is primarily due to the overproduction of bilirubin, which means a higher number of red blood cells are being destroyed, leading to an increased concentration of bilirubin in the blood. Additionally, there is a metabolic disorder of bilirubin in the child. The liver cells' ability to take up and bind bilirubin decreases, which can cause an increase in the concentration of unbound bilirubin in the serum, resulting in jaundice. This mainly occurs in cases of hypoxia, infection, and some congenital diseases, among others. Certain medications and reduced thyroid function can also present this symptom. The third issue is an impairment in the bile's ability to excrete bilirubin, which can occur when the child has neonatal hepatitis or congenital metabolic defects, leading to jaundice.