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Ventricular septal defect
Is ventricular septal defect related to premature birth?
Generally, premature birth is not related to ventricular septal defect. Although a premature infant is born early, their heart structure, lungs, and other body structures are normal. Ventricular septal defect is often caused by a developmental disorder of the septum during the fetal period, manifesting as left-to-right or right-to-left shunting. Generally, patients with a simple ventricular septal defect can survive normally. They can be completely cured through interventional treatment or surgical surgery, so there is no need to worry excessively. There is no direct connection between ventricular septal defect and premature birth.
Is congenital heart disease with perimembranous ventricular septal defect serious?
Simple congenital heart defects, such as perimembranous defects, generally are not particularly severe. However, there are several scenarios to consider: The first scenario involves a large defect that typical occluders cannot address; in such cases, surgical intervention is required. This type of defect is generally considered severe. Another scenario is when a cardiac defect has been present for an extended period, leading to repeated shortness of breath, heart failure, cardiac enlargement, and even pulmonary arterial hypertension. This situation would be considered a severe ventricular septal defect, caused by the failure to treat the membranous part of the septal defect in time. Thus, the majority of ventricular septal defects are not very severe in the early stages. They become serious if the defect is too large or if there is coexistence with other defects, such as an overriding aorta or poorly developed pulmonary artery, among other complex congenital heart diseases. Regardless, patients with perimembranous congenital heart defects should undergo surgical treatment early to avoid missing the opportunity for surgery.
Can someone with a ventricular septal defect travel by airplane?
Generally, individuals with ventricular septal defects (VSD) with no complications do not display symptoms and are able to travel by airplane. When such defects are at an early stage, where cardiac function has not been significantly impaired and there is left-to-right shunting without heart failure or pulmonary hypertension, flying is generally tolerated. After surgery for a ventricular septal defect, patients usually can fly. However, if the patient with a ventricular septal defect is older and in the advanced stages of the condition, exhibiting clear symptoms of heart failure such as difficulty breathing and chest tightness with significant exertional stress, such patients are unable to lie down and should not fly. Additionally, if there is right-to-left shunting or severe pulmonary hypertension has developed, it is unsafe for them to fly as they could potentially face emergencies during the flight.
Can a ventricular septal defect heal by itself?
Some ventricular septal defects can heal on their own. For ventricular septal defects smaller than five millimeters, some patients can heal on their own; the defects that generally heal are those in the membranous and muscular parts of the septum, and the younger the age, the greater the likelihood of healing. If the ventricular septal defect still exists after the age of five, it generally cannot heal on its own. For ventricular septal defects that do not heal spontaneously, if they significantly affect hemodynamics, interventional treatment or surgical surgery is needed for repair. If the impact of the ventricular septal defect on hemodynamics is not significant, regular follow-up examinations can be conducted to understand the condition of the disease.
What would happen if the ventricular septal defect occluder dislodges?
An occluder generally works like two umbrella surfaces clamped over the orifice of the interventricular septal defect, achieving the method whereby tissue blood flow enters from the left side of the septum to the right side. Once an occluder dislodges, it can lead to a dangerous situation, as the occluder has a membrane that can easily get caught on the tendons of the mitral valve, causing severe mitral regurgitation; it can also get stuck at the mitral valve orifice, obstructing the outflow of blood; it may also lead to aortic valve insufficiency; if it gets caught in the major arteries, it can cause arterial embolism, leading to sudden death in the patient. Therefore, the dislodgement of an occluder is a very dangerous phenomenon.
What are the complications of ventricular septal defect?
The complications of ventricular septal defect mainly include the following: 1. Causes infective endocarditis, with the highest incidence occurring between the ages of 15 and 29; 2. Leads to aortic valve insufficiency; 3. Causes conductive blockages; 4. Leads to heart failure, and can even lead to Eisenmenger syndrome. Therefore, patients with ventricular septal defects should actively complete examinations such as echocardiography. If there are indications for surgery, they should promptly visit a cardiac surgeon for active surgical treatment to correct the anatomical abnormalities of the ventricular septal defect and prevent the condition from worsening and leading to serious consequences.
Does interventricular septal defect occasionally cause chest pain?
Ventricular septal defect is a type of congenital heart disease, occurring when the ventricles of the heart develop poorly during the embryonic stage, leading to a defect in the ventricular septum. This often results in a left-to-right shunt which affects the efficiency of the heart's function. In severe cases, it can cause a right-to-left shunt. Generally, these conditions do not affect the coronary circulation or cause chest pain in patients. However, some patients may develop endocarditis at the ventricular septum, which sometimes leads to the formation of vegetations on the cardiac valves or blood clots on the abdominal wall. If thrombosis or vegetations occur, there is a possibility of these breaking off. If they enter the coronary arteries, they can cause chest pain. However, such events are extremely rare, with an occurrence rate of less than one in a thousand.
Symptoms of ventricular septal defect
The main symptoms of a ventricular septal defect depend on the size of the defect and the age of the patient. Generally, smaller ventricular septal defects, such as those measuring only one to two millimeters, result in very little shunting from the left ventricular system to the right ventricular system. This is akin to a tiny amount of air seeping through a crack in a window or door. In such cases, the patient typically does not exhibit noticeable symptoms. However, if the defect is larger, over five millimeters, a significant amount of blood passes from the left ventricular system into the right ventricular system, which can cause substantial congestion in the right or both lungs of the right ventricular system, or lead to recurrent respiratory infections and congestive heart failure. The affected individual may show disinterest in eating, feeding difficulties, and delayed development. As the patient grows older, they may exhibit reduced activity endurance, fatigue or palpitations, and shortness of breath. Gradually, cyanosis and right heart failure may develop. Additionally, patients are at risk of developing infective endocarditis, fever without symptoms, and recurring fever.
Is perimembranous ventricular septal defect serious?
The ventricles are separated by the interventricular septum, which divides the left and right ventricles. Typically, after blood is ejected from the left ventricle and oxygenated by tissues, it returns to the right ventricle. The blood from the right ventricle gets oxygenated through the pulmonary artery before it can return to the left ventricle. These two should be isolated from each other before passing through the lungs. The interventricular septum consists of the membranous and muscular parts, and a defect in either part can affect oxygenation. Therefore, whether the defect is in the membranous or muscular portion is not an indicator for diagnosing the severity of the interventricular septum defect; rather, the size of the defect should be considered. Generally speaking, a defect of about 10mm in the interventricular septum, especially when accompanied by other conditions such as Tetralogy of Fallot, is considered severe. Moreover, when the defect reaches over 10mm, surgical intervention is required as minimally invasive surgery would not be feasible; these are considered severe interventricular septum defects. Also, there are cases with multiple complex defects or larger defects that should be surgically treated as soon as possible.
Can ventricular septal defect avoid amniocentesis?
Because ventricular septal defect is a type of congenital heart disease, it is recommended that patients adopt a light diet and especially avoid spicy and stimulating foods due to the condition of ventricular septal defects. It is still recommended to pursue active and early surgical treatment to improve symptoms. If there is poor cardiac function, or symptoms of palpitations and chest tightness occur, amniocentesis should not be performed as it carries certain risks. However, for milder symptoms that can be timely controlled, amniocentesis and other tests may be considered, but it is also necessary to rule out other diseases. If the patient does not have any special diseases in the past, such as coronary heart disease or diabetes, the procedure can be handled.