Does a ventricular septal defect easily cause choking on milk?

Written by Di Zhi Yong
Cardiology
Updated on December 21, 2024
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Ventricular septal defect can lead to choking on milk, as it can cause pathological murmurs in children, especially prominent in the chest area, which is a type of congenital heart disease. Currently, early surgical treatment is recommended for these children. The choking on milk might be due to reasons related to the digestive system, but it is still somewhat related to the ventricular septal defect. It is also advised to eat smaller, more frequent meals to avoid choking or aspiration, which can lead to bronchitis or recurrent respiratory infections.

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Written by Di Zhi Yong
Cardiology
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Can girls with ventricular septal defect have children?

Because ventricular septal defect is a type of congenital heart disease, it is currently recommended that patients undergo surgical treatment. With active treatment, this disease can be cured. Once cured, the patient's symptoms will subside, and no murmur can be heard in the precordial area, and it is possible to choose to have children in this situation. If this condition is not dealt with promptly, it is not recommended to have children. This is because it can cause an excessive load on the heart, especially when the blood volume increases, leading to occurrences of ischemia and hypoxia, which are very dangerous. If treated, especially within a year after surgery, it is possible to consider having children, but there are certain risks.

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Written by Xie Zhi Hong
Cardiology
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Will being overweight affect the surgery for a ventricular septal defect?

Ventricular septal defect surgery is performed by puncturing the right femoral vein, guiding the catheter into the inferior vena cava, through the atria and ventricles, and reaching the defect via the right ventricle. The occluder can then be delivered through the catheter into the left ventricle, where the umbrella is opened, followed by opening it in the right ventricle; this describes the surgical process. Therefore, generally speaking, the surgical process is not affected in most patients who are obese. However, severe obesity can impact the accuracy of vascular localization during puncture by the surgeon. Secondly, if complications such as anesthesia issues or other emergencies arise during the surgery, severely obese patients may experience a reduced success rate in emergency surgical interventions, hence it is recommended that patients control their weight. Furthermore, severe obesity can exacerbate the cardiac load originally induced by the ventricular septal defect, thus affecting the surgical tolerance in patients whose cardiac function is already compromised by the defect. It is recommended that these patients control their weight.

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Written by Zhang Zhi Gong
Cardiothoracic Surgery
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Symptoms of ventricular septal defect

The main symptoms of a ventricular septal defect depend on the size of the defect and the age of the patient. Generally, smaller ventricular septal defects, such as those measuring only one to two millimeters, result in very little shunting from the left ventricular system to the right ventricular system. This is akin to a tiny amount of air seeping through a crack in a window or door. In such cases, the patient typically does not exhibit noticeable symptoms. However, if the defect is larger, over five millimeters, a significant amount of blood passes from the left ventricular system into the right ventricular system, which can cause substantial congestion in the right or both lungs of the right ventricular system, or lead to recurrent respiratory infections and congestive heart failure. The affected individual may show disinterest in eating, feeding difficulties, and delayed development. As the patient grows older, they may exhibit reduced activity endurance, fatigue or palpitations, and shortness of breath. Gradually, cyanosis and right heart failure may develop. Additionally, patients are at risk of developing infective endocarditis, fever without symptoms, and recurring fever.

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Written by Xie Zhi Hong
Cardiology
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Is congenital heart disease with perimembranous ventricular septal defect serious?

Simple congenital heart defects, such as perimembranous defects, generally are not particularly severe. However, there are several scenarios to consider: The first scenario involves a large defect that typical occluders cannot address; in such cases, surgical intervention is required. This type of defect is generally considered severe. Another scenario is when a cardiac defect has been present for an extended period, leading to repeated shortness of breath, heart failure, cardiac enlargement, and even pulmonary arterial hypertension. This situation would be considered a severe ventricular septal defect, caused by the failure to treat the membranous part of the septal defect in time. Thus, the majority of ventricular septal defects are not very severe in the early stages. They become serious if the defect is too large or if there is coexistence with other defects, such as an overriding aorta or poorly developed pulmonary artery, among other complex congenital heart diseases. Regardless, patients with perimembranous congenital heart defects should undergo surgical treatment early to avoid missing the opportunity for surgery.

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Written by Di Zhi Yong
Cardiology
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Can ventricular septal defect be detected during prenatal screening?

Ventricular septal defect is a type of congenital heart disease that can only be diagnosed through an echocardiogram. Routine prenatal screenings cannot detect it. Particularly for newborns, it’s advisable to have an echocardiogram, and for fetuses, a four-dimensional ultrasound is recommended. Sometimes, ultrasound results can only serve as a reference and may not indicate actual problems, but there is also a certain margin of error. If a child has congenital heart disease, it is advisable to have regular echocardiogram check-ups at the hospital. This allows timely identification of the cause, and determination of whether surgery is necessary for treatment, which is the ultimate goal.