Cerebral infarction (Stroke)


Is a brain infarction dangerous?
The risk associated with a cerebral infarction primarily depends on the extent and location of the infarction. If the infarction is small and located in less critical areas of brain function such as the frontal or temporal lobes, the symptoms may not be severe and might only include dizziness and sleepiness. If the infarction is extensive and occurs in crucial areas such as the basal ganglia, thalamus, or brainstem, the symptoms can be very severe. This could potentially lead to hemiplegia, aphasia, and even coma or death.


Can cerebral infarction be cured completely?
Stroke is not a disease that can be completely cured. Instead, the goal of treatment is to prevent further occurrences and developments of stroke, and there is also the potential for some relative improvement later on. This is considered a very good treatment goal. In the case of stroke, it involves the ischemic necrosis of nerve cells. Additionally, nerve cells have a characteristic that once they die, they cannot regenerate. Therefore, in clinical practice, there are no treatments available that can promote the regeneration of nerves. Treatment can only compensate for the functions of the surrounding healthy nerves to take over the functions of the dead nerves. However, this compensatory process is very slow, often taking two to three months to show significant progress. Hence, it is necessary to continue active rehabilitation exercises later to promote the formation of this compensatory mechanism.


What is a cerebral infarction?
Cerebral infarction is essentially a manifestation of ischemic necrosis of nerve cells. However, in clinical practice, there are many factors that can cause cerebral infarction. It is essential to treat the underlying causes to effectively prevent the further occurrence and progression of cerebral infarction. One characteristic of nerve cells is that once necrosis occurs, they cannot regenerate. Consequently, all patients who suffer from cerebral infarction may have some residual neurological deficits. Mild symptoms include headaches and dizziness, while severe cases may experience numbness and weakness in the limbs, speech impairments, and even more severe conditions such as a vegetative state or death. Therefore, in the case of cerebral infarction, it is crucial to focus on prevention early on to reduce the extent of necrosis. This can effectively ensure one's safety and reduce the occurrence of various complications and sequelae.


Is it good to drink milk after a stroke?
Patients with cerebral infarction can drink milk, but it must be analyzed according to the specific condition at the time because patients with cerebral infarction often experience coma and accompanying dysphagia with coughing. In such cases, drinking milk is not a problem, but attention must be paid to changes in dietary habits. If necessary, a gastric tube must be inserted through the nose into the stomach for feeding to avoid direct oral intake, which can cause coughing, aspiration, and lead to pneumonia. Therefore, this point must be carefully considered. For ordinary people in daily life, a cup of milk a day is a very good choice because milk mainly contains protein and does not contain a lot of fat and sugar. For patients with cerebral infarction, this diet is very ideal. If fresh milk can be used in everyday life, it is more beneficial for promoting the patient's recovery.


post-stroke sequelae
Brain infarction is a very common type of ischemic cerebrovascular disease, which mainly includes the following types: the most common are lacunar infarction, cerebral thrombosis, and cerebral embolism. The neurological sequelae of lacunar infarction are not particularly typical, and patients may exhibit mild incomplete paralysis of limbs, decreased muscle strength, reduced sensation, or ataxia. Cerebral thrombosis and cerebral embolism often cause extensive brain infarction, where patients typically experience severe limb paralysis, loss of sensation, increased tendon reflexes, positive pathological reflexes, and an inability to care for themselves.


Is a cerebral infarction serious?
Stroke is a very serious disease; its essence lies in the ischemic necrosis of nerve cells. A characteristic of nerve cells is that once they undergo ischemic necrosis, they cannot regenerate. Along with cardiac muscle cells, they are the only cells in the human body that cannot regenerate once they have died. Therefore, for the part of the tissue where nerve cells have already died, such as in cases of cerebral thrombosis or stroke where the tissue has already been affected, it is impossible to completely restore it. If the affected area is relatively small, some compensation might be possible. However, if the affected area is relatively large, this will lead to a significant amount of ischemic necrosis in the neural tissue, which can cause total dysfunction of various tissue structures in the body, and in more severe cases, can lead to coma and eventually the death of the patient. Therefore, it is imperative to take this seriously in clinical practice.


Massage methods for cerebral infarction
For patients with cerebral infarction, many of them suffer from facial paralysis, often characterized by crooked mouth and eyes and drooling. To alleviate symptoms and help improve the asymmetry of the mouth and eyes, it is suggested to massage the facial and neck muscles of the patient. It is generally recommended to seek an experienced Traditional Chinese Medicine practitioner who can use manual massage techniques, targeting specific acupoints to effectively relieve pain and improve symptoms. For patients with motor dysfunction or even hemiplegia, manual massage can effectively help prevent muscle atrophy, relieve muscle rigidity, and reduce muscle tone.


Can the blood vessels in a cerebral infarction be unblocked?
Whether the blood vessels in a cerebral infarction can be reopened is closely related to time. If it is within the hyperacute phase, which means within six hours from the onset of clinical symptoms to hospital admission, the majority of cases can undergo thrombolysis intravenously, or arterial thrombectomy to reopen the blood vessels. However, if the time exceeds this window, reopening is no longer feasible, and forced reopening at this stage can easily lead to the detachment of distal thrombi or cause reperfusion injury, which is more detrimental to the patient's condition. Therefore, it is impossible to reopen the blood vessels during the chronic phase or non-hyperacute phase. If treatment of this vascular occlusion is desired, vascular anastomosis can be considered, though the specific type of anastomosis surgery depends on the patient's specific condition at the time. Thus, the possibility of reopening a blood vessel in a cerebral infarction certainly depends on timing.


Can a cerebral infarction be cured?
Stroke is the most common cerebrovascular disease. It occurs when the arteries supplying blood to the brain become blocked, leading to ischemic necrosis of the brain tissue in that region. This results in a series of neurological deficits. Theoretically, once a stroke has occurred, it cannot be completely cured because brain cells are non-regenerative. Once they die, they cannot be revived, and the function of the neurons in that area is completely lost. However, the significance of acute phase treatment lies in the rescue of the ischemic penumbra. The function of these cells can be restored after treatment, and thereafter, they may compensate for the function of the central necrotic area. Thus, patients who receive treatment may regain some neurological functions. Another condition is that the patient must be brought to the hospital within 4.5 hours of the onset of symptoms. If there are no clear contraindications, RT-PA intravenous thrombolytic treatment can be administered. The success rate of this thrombolysis is relatively high, and about 30% of patients may be fully cured without any sequelae.


Precursors to cerebral infarction
For patients with cerebral infarction, before the onset of the illness, they often lack specific prodromal symptoms. Some patients may experience mild headaches, dizziness, nausea, and a clear feeling of discomfort. Additionally, some patients might experience numbness in the legs and involuntary twitching of facial muscles before the onset. Subsequently, some patients may also start to have drooping of the corner of the mouth to one side, drooling, slurred speech, and distinct articulation disorders. When these clinical presentations occur, it is necessary to be vigilant and seek timely medical attention at a local hospital. A cranial CT or MRI should be performed to confirm the diagnosis.