Subscribe
68
Wei Shi Liang
Intensive Care Unit
About me
Graduated from Shanxi Medical University with a degree in Clinical Medicine in 2006, and has been working in the field of Critical Care Medicine ever since.
Proficient in diseases
Treatment of severe infections, ARDS, severe trauma, MODS, and other diseases.
Voices
Can bronchial asthma be treated with nebulization?
Patients with bronchial asthma can undergo nebulization, especially pediatric asthma patients who are more suited for nebulizer therapy. Nebulization inhalation is currently a safe and effective method for treating childhood asthma and is listed by the World Health Organization as the first choice for global asthma prevention and treatment. For severe asthma during acute exacerbations and attack periods, nebulization therapy is not recommended. When using nebulization therapy, it is important to regularly disinfect the nebulizer and replace it periodically. During severe bronchospasm, it is important to avoid using excessive doses of receptor agonists, and care should be taken to avoid too low drug concentrations during nebulization, as it is not conducive to the effectiveness of the treatment.
Symptoms and signs of subarachnoid hemorrhage
The typical symptoms of subarachnoid hemorrhage include headache, vomiting, and sudden severe pain, accompanied by vomiting, pale complexion, and whole-body cold sweats. Additionally, there may be disturbances in consciousness and psychiatric symptoms. Most patients do not experience disturbances in consciousness, but may exhibit restlessness and agitation. Severe cases can show varying degrees of clouded consciousness, and even coma, with a few instances displaying seizures and psychiatric symptoms. Furthermore, signs of meningeal irritation are also present, particularly common and pronounced in young and middle-aged patients, characterized by neck stiffness, headaches, and vomiting.
Can severe pancreatitis be cured?
Severe pancreatitis can be cured, but because its complications are severe, it may be life-threatening. Severe pancreatitis is caused by a variety of etiologies leading to local inflammation, necrosis, and infection of the pancreas, accompanied by systemic inflammatory responses and persistent organ failure. Currently, comprehensive treatment for severe pancreatitis has become very mature, but its mortality rate is still as high as 17%. Currently, with a deeper understanding of the pathology, physiology, and disease progression of severe pancreatitis, there have been advances in treatment modalities, treatment concepts, and means of organ function support for severe pancreatitis. However, the mortality rate for severe pancreatitis remains high, though it can still be cured.
Two major signs of severe pancreatitis
In patients with severe pancreatitis, physical examination may reveal abdominal distension with tympanic percussion sounds, prominent tenderness in the upper middle abdomen, and potentially widespread abdominal pain centered in the upper middle area. Some may exhibit rebound tenderness, moderate muscle tension is common, and a few cases may demonstrate shifting dullness. Occasionally, a mass in the upper middle abdomen can be palpated, possibly due to fluid in the lesser sac. Auscultation may reveal diminished or absent bowel sounds, accompanied by cessation of passing gas or stool, indicating features of paralytic ileus.
Symptoms of severe pancreatitis
The main symptom of severe pancreatitis is abdominal pain. This type of abdominal pain manifests as intense pain in the upper-middle abdomen, which radiates to the back and both sides of the body. The pain is widespread and severe, with about 95% of patients experiencing abdominal pain. Onset often occurs following binge eating or excessive drinking, and the pain worsens after eating. Another symptom is abdominal distension, which is also a common symptom. It is caused by extensive effusion in the abdominal cavity and retroperitoneum, as well as intestinal paralysis. Fever in the early stage of the disease is also a common symptom, resulting from the absorption of a large amount of necrotic tissue. Fever occurring in the later stages is often caused by infections triggered within the abdominal cavity.
Prevention and Treatment of Bronchial Asthma
The prevention and treatment of bronchial asthma mainly involve the following aspects: First, eliminating the causes and triggers of the disease; second, preventing diseases and pests such as allergic rhinitis and gastroesophageal reflux disease; third, immunomodulation; fourth, routinely checking if inhaled medications are used correctly; fifth, education and management of asthma patients, mainly allowing them through long-term, appropriate, and sufficient treatment to fully and effectively control asthma attacks. Additionally, understanding individual differences in asthma triggers to avoid occurrences, learning patient self-monitoring of disease progression, mastering the use of inhalers and peak flow meters, and educating patients on simple self-help methods are essential. It's important for patients and doctors to jointly develop a plan to prevent asthma attacks and maintain long-term stability. These are the basic elements of the prevention and treatment of bronchial asthma.
Why does hyperkalemia cause acidosis?
The concentration of potassium ions in serum is 3.5 to 5.5 millimoles per liter, and concentrations above 5.5 millimoles per liter are considered hyperkalemia. In the state of hyperkalemia, potassium ions in the extracellular fluid move into the intracellular fluid, while hydrogen ions in the intracellular fluid move to the extracellular fluid. At this time, through a compensatory mechanism, there is an increase in hydrogen ions in the extracellular fluid, significantly higher than normal levels, resulting in acidosis. Therefore, hyperkalemia often accompanies metabolic acidosis, which in turn affects the renal tubular epithelial cells, causing an abnormal alkaline urine. This is the main reason why hyperkalemia leads to acidosis.
Is hyperkalemia acidosis?
Hyperkalemia is not acidosis, but during acidosis, the hydrogen ions of the gastric fluid within cells enter the cells, causing the potassium ions inside the cells to move to the extracellular fluid, resulting in hyperkalemia. Clinically, it is commonly seen in organic acidosis, lactic acidosis, diabetic ketoacidosis, and acute renal failure causing acidosis. Once hyperkalemia occurs and is diagnosed, immediate treatment should be administered. First, the primary disease should be treated; next, serum potassium should be reduced. In particularly severe cases, bedside hemofiltration can be administered, and the cardiotoxic effects of hyperkalemia should be mitigated.
Can a pulmonary embolism be detected by a CT scan?
CT can detect acute pulmonary embolism, using CT angiography, which is the preferred imaging method for diagnosing pulmonary embolisms. It mainly shows central filling defects, as well as eccentric and abdominal wall filling defects, and complete filling defects. A diagnosis of pulmonary embolism can be made through the visualization of these filling defects on a CT scan. Additionally, some indirect signs, such as pulmonary hypertension, right ventricular enlargement, and the common mosaic sign, can also be observed. These are characteristic CT manifestations of acute pulmonary embolism.
Severe pancreatitis is what disease
Acute severe pancreatitis is a disease caused by various etiologies, characterized by local necrosis, inflammation, and infection of the pancreas, accompanied by systemic inflammatory response and persistent organ failure. The current mortality rate is still as high as 17%. The course of acute severe pancreatitis can generally be divided into three periods. First, the acute response period, occurring up to about two weeks after onset, is characterized by a systemic inflammatory response. Second, the systemic infection period, from two weeks to about two months, is characterized by infection of pancreatic or peripancreatic necrosis. Third, the residual infection period, occurring two to three months later, where the main clinical manifestation is systemic malnutrition.