Subscribe
56
Chen Xie
Endocrinology
About me
Loudi Central Hospital, Endocrinology Department, attending physician, has been engaged in clinical work in endocrinology for many years, with rich clinical experience in the diagnosis and treatment of endocrine system diseases.
Proficient in diseases
Specialize in common diseases related to the hypothalamus, pituitary gland, thyroid gland, parathyroid glands, adrenal glands, and the reproductive axis.
Voices
What should be paid attention to regarding thyroid cysts?
Thyroid cysts generally do not show obvious symptoms. When a cyst significantly enlarges or internal bleeding occurs, compressive symptoms or pain may appear. Thyroid cysts can lead to hyperthyroidism, and if persistent, may eventually cause hypothyroidism. Therefore, it is necessary to regularly monitor thyroid function to understand the status of thyroid activity. The treatment of thyroid cysts primarily involves distinguishing whether they are malignant. For thyroid cysts suspected to be malignant, surgical treatment is required. Generally, thyroid cysts are benign and only require regular observation and follow-up.
Can Hashimoto's thyroiditis be cured?
Hashimoto's thyroiditis is an autoimmune thyroiditis where the body itself has autoantibodies that lead to the destruction of thyroid follicular cells, resulting in the failure of thyroid cells. Therefore, Hashimoto's thyroiditis can manifest as hyperthyroidism, euthyroidism, and hypothyroidism. Ultimately, Hashimoto's thyroiditis may develop into hypothyroidism. Hypothyroidism is proliferative and generally cannot be completely cured, but clinical remission can be achieved through oral administration of levothyroxine. However, Hashimoto's thyroiditis cannot be completely cured and requires lifelong replacement therapy.
Can Hashimoto's thyroiditis be cured?
Hashimoto's thyroiditis is an autoimmune thyroiditis in which the body itself has autoimmune antibodies, leading to the destruction of thyroid follicular cells and the release of a large amount of thyroid hormones, possibly resulting in hyperthyroidism. Some patients may experience hyperthyroidism for years or even decades, which requires anti-hyperthyroidism treatment. However, as the disease progresses, further destruction of thyroid function may occur, resulting in hypothyroidism. For patients with Hashimoto's thyroiditis, once hypothyroidism occurs, it is usually progressive. Therefore, once hypothyroidism occurs in patients with Hashimoto's thyroiditis, lifelong replacement therapy with levothyroxine is necessary, as Hashimoto's thyroiditis cannot be completely cured, but clinical remission can be achieved through replacement therapy.
What is the heart rate for Hashimoto's thyroiditis?
Hashimoto's thyroiditis is an autoimmune thyroid disease. The course of the disease can include hyperthyroidism, euthyroidism, and hypothyroidism. When Hashimoto's thyroiditis is at different stages of thyroid function, the changes in heart rate are also inconsistent. When Hashimoto's thyroiditis coincides with hyperthyroidism, the heart rate may be fast, even exceeding 100bpm. When Hashimoto's thyroiditis coincides with normal thyroid function, the heart rate may be within the normal range, around 60bpm to 100bpm. When Hashimoto's thyroiditis coincides with hypothyroidism, the heart rate may slow down, possibly dropping below 60bpm. Therefore, in Hashimoto's thyroiditis, as the thyroid function varies, so does the heart rate.
Does Hashimoto's thyroiditis require medication?
Hashimoto's thyroiditis refers to the destruction of thyroid tissue by autoimmune cells. Therefore, during the course of the disease, three states of thyroid function can be manifested: hyperthyroidism, euthyroid state, and hypothyroidism. When the patient presents with hyperthyroidism, it is possible to treat with anti-thyroid drugs, but the dosage of the anti-thyroid drugs should be reduced. When thyroid function is normal, no special treatment is needed, and regular thyroid function testing is sufficient. When there is concurrent hypothyroidism, thyroid hormone replacement therapy is required to achieve clinical healing.
How is hyperuricemia treated?
The treatment of hyperuricemia mainly includes: First, improving lifestyle, including healthy eating with a low-purine diet, quitting smoking, drinking more water, exercising regularly, and controlling weight. Second, alkalinizing the urine using sodium bicarbonate to maintain urine pH between 6.2 and 6.9, which facilitates the excretion of uric acid. Third, avoiding medications that increase blood uric acid levels, such as diuretics, corticosteroids, and insulin, among others. Fourth, using medications that lower uric acid, including drugs that increase uric acid excretion, mainly benzbromarone and probenecid, and drugs that inhibit uric acid synthesis, such as allopurinol and febuxostat. The choice of medication has specific indications, contraindications, and side effects, and it is advised to use these medications under the guidance of a specialist and not to self-medicate.
What is contraindicated for hyperuricemia?
Hyperuricemia contraindications include the following points: 1. Strictly limit the intake of purines, restrict foods that contain more purines, including animal organs, bone marrow, seafood, fermented foods, beans, etc. 2. Reduce the intake of fats, as fats can decrease the excretion of uric acid. 3. Avoid high-protein foods, such as lean meat, chicken, and duck, which should be boiled and the broth discarded before consumption to avoid eating stewed or marinated meats. 4. Abstain from alcohol, as alcohol can lead to the accumulation of lactate in the body and inhibit the excretion of uric acid, easily triggering gout attacks. 5. Avoid hot pot, as the main ingredients in hot pot are animal organs, shrimp, shellfish, and seafood, and drinking beer with it can aggravate the condition, making these items contraindications for hyperuricemia.
Is the ESR high in Hashimoto's thyroiditis?
Hashimoto's thyroiditis belongs to autoimmune thyroiditis, which is caused by the presence of autoantibodies that destroy the thyroid follicular cells, leading to the failure of thyroid cells. For patients with Hashimoto's thyroiditis, the erythrocyte sedimentation rate (ESR) generally does not increase. However, if a patient experiences neck discomfort or sore throat, and the ESR increases, even accompanied by fever, it is necessary to rule out whether subacute thyroiditis is present. If the patient has subacute thyroiditis, it indicates that a viral infection has caused the destruction of the thyroid cells, which might increase the ESR.
Can hyperuricemia be cured?
Hyperuricemia is a metabolic disease caused by disorders in purine metabolism and is clinically divided into primary and secondary categories. Primary hyperuricemia is caused by congenital abnormalities in purine metabolism and is often associated with obesity, dyslipidemia, hypertension, atherosclerosis, and coronary heart disease. Secondary hyperuricemia, on the other hand, is caused by certain systemic diseases or medications. Therefore, whether hyperuricemia can be cured depends on the underlying condition, and the efficacy of treatment cannot be generalized. For hyperuricemia caused by medications or systemic diseases, it can be cured by treating the disease or discontinuing the medication. However, for hyperuricemia caused by congenital abnormalities in purine metabolism, it can be clinically managed by controlling diet and medication, but it is not curable.
Steps for Gestational Diabetes Testing
The screening steps for gestational diabetes involve, for most women, the recommendation to undergo a 75-gram glucose tolerance test at the hospital between 24 to 28 weeks of pregnancy. The method includes fasting blood draw, consuming 75 grams of glucose, and monitoring blood sugar levels one hour and two hours after glucose intake, to determine the presence of gestational diabetes. However, for pregnant women at high risk for diabetes, it is recommended to complete the 75-gram glucose tolerance test early in pregnancy to detect gestational diabetes early and initiate treatment promptly to reduce the risks to both the fetus and the mother.