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Li Jiao Yan
Neonatology
About me
Li Jiaoyan, female, attending physician, Master of Pediatric Medicine integrating Chinese and Western medicine. Graduated from Hunan University of Chinese Medicine.
Research interests include integrating Chinese and Western medicine for neonates and pediatric internal medicine, with expertise in treating common and frequent diseases in neonates and children, as well as emergency rescue of critical conditions. Proficient in monitoring the growth and development of children in early stages, assessing their developmental levels, and providing guidance.
Proficient in diseases
Treatment of common and multiple diseases in newborns and children, as well as emergency treatment of critical illnesses, monitoring of early childhood growth and development, assessment of early childhood developmental levels, and guidance.
Voices
What is considered high for neonatal jaundice?
Neonatal jaundice is common during the neonatal period and can be divided into physiological jaundice and pathological jaundice. Physiological jaundice does not require intervention or treatment, and the baby's condition is generally okay. For physiological jaundice, the bilirubin level typically does not exceed 6 within the first 24 hours after birth, then not exceeding 9 from 24 to 48 hours, not exceeding 12 from 48 to 72 hours, and not exceeding 15 after 72 hours. If the monitored bilirubin levels exceed these ranges, there may be pathological jaundice, indicating abnormally high levels.
Newborn jaundice can be divided into several types.
Neonatal jaundice is one of the common symptoms during the neonatal period, especially in early newborns. It can be a symptom of the normal developmental process of newborns, or it can be a manifestation of certain diseases. Neonatal jaundice is relatively common in newborns, with about 80% of full-term infants visibly showing jaundice. Jaundice is classified into physiological jaundice and pathological jaundice. If it is pathological jaundice, there are different classifications. Based on different characteristics of bilirubin elevation, it is divided into high direct bilirubinemia and high indirect bilirubinemia. Physiological jaundice does not require intervention. However, if it is pathological jaundice, it is necessary to clarify its classification and determine which type it belongs to. Generally, if it is pathological jaundice, timely intervention and treatment are needed. If untreated, and if the jaundice level significantly increases, it may lead to serious complications.
Rickets is a disease that affects bone development in children due to vitamin D deficiency, calcium, or phosphate. It is treatable with proper nutrition and supplementation.
Rickets, also known as vitamin D deficiency rickets, is caused by a lack of vitamin D, leading to abnormal calcium and phosphorus metabolism in the body. The calcium salts cannot be properly deposited in the growing parts of the skeleton, causing incomplete mineralization of the bone tissue during growth. This results in a chronic systemic nutritional disease closely related to lifestyle, characterized by skeletal abnormalities. The most common is nutritional vitamin D deficiency, which is self-limiting. Typically, sufficient vitamin D supplementation and appropriate sun exposure can cure it. If caused by other severe diseases, vitamin D deficiency will significantly manifest liver and kidney function abnormalities. Additionally, some congenital genetic diseases make treatment more difficult when vitamin D levels are deficient.
Does Kawasaki disease affect lifespan?
Kawasaki disease, also known as mucocutaneous lymph node syndrome, has an unclear pathogenesis. Its pathological changes primarily involve a systemic circulatory disease characterized by vasculitis. A severe complication can lead to cardiac lesions. Generally, Kawasaki disease is self-limiting and most cases have a good prognosis. However, if Kawasaki disease is not effectively treated and results in associated coronary artery aneurysms or large artery aneurysms, it could lead to acquired heart disease. Such cardiac issues could potentially affect cardiac function later on. This might impact lifespan, but if Kawasaki disease is treated aggressively and effectively, the prognosis is good and the impact on future health is minimal.
Is rickets easy to treat?
Rickets is primarily a nutritional disease caused by insufficient levels of vitamin D leading to disturbances in calcium and phosphate metabolism, resulting in abnormal bone development. The most common type is nutritional vitamin D-deficiency rickets. This type of disease is self-limiting and thus relatively easy to treat. It requires early detection and proactive intervention. Early measures include encouraging children to engage in outdoor activities and supplementing with appropriate doses of vitamin D, which can effectively treat rickets. However, there are also special causes of vitamin D deficiency such as significant anomalies in liver or kidney function and certain genetic diseases that severely impair vitamin D absorption. When vitamin D treatment is ineffective, it may be due to other special causes. Treatment of rickets under these circumstances can be more challenging, and genetic diseases may respond poorly to treatment.
What are the symptoms of rickets?
The condition generally referred to as rickets is most commonly nutritional vitamin D deficiency rickets, a type of nutritional disease that is prevalent among infants and young children, particularly very young babies. It primarily manifests as changes in the bones where growth is most rapid and can also affect muscle development and changes in nervous excitability. Initially, especially within the first three months, symptoms in very young infants typically include increased nervous excitability, such as crying easily, excessive head sweating, and as a result of the sweating, frequent head shaking, irritability, restlessness, and noticeable crying and restlessness at night. These symptoms at this stage are not necessarily indicative of typical rickets. As the condition progresses, changes in the skeleton gradually become apparent. In infants under six months with rickets, the changes are mainly in the skull, typically feeling like pressing on a ping-pong ball. After six months of age, the head shape becomes squared or box-like, and the head circumference is larger than normal. Later, the characteristic beading changes of rickets appear, with beaded protrusions above the ribs, followed by the formation of blunt, circular raised areas at the wrists and ankles, referred to as wrist and ankle bracelets. Around the age of one, infants may develop a deformity resembling pigeon chest. As the child begins to stand and walk, the weight bearing on the lower limbs may lead to deformities, such as bowlegs or knock-knees, and sometimes even K-shaped deformities of the legs, and some might develop spinal deformities.
Neonatal jaundice bath Chinese medicine
Neonatal jaundice is called "fetal jaundice" in Traditional Chinese Medicine (TCM). It primarily refers to infants born with jaundice as the main symptom, characterized by yellowing of the skin and facial area. This condition is related to prenatal constitution, hence it is also known as fetal jaundice or fetal dyspepsia. The causes of fetal jaundice are divided into internal and external factors. Internal factors involve the fetus being affected by the toxins of damp-heat or the evil of deficient-cold and damp-cold from the mother. External factors mainly include the baby being affected by damp-heat or cold-damp evils during or after birth, with damp-heat evils being more common. The main affected organs are the liver, gallbladder, spleen, and stomach, with the key pathogenic mechanism being prenatal damp retention. Commonly used Chinese herbal baths for neonatal jaundice, particularly suitable for damp-heat type jaundice, are generally selected based on the specific conditions of the child, adjusting formulas like Yinchenhao Tang to tailor to the exact pattern of the baby.
Kawasaki disease peeling symptoms
Kawasaki disease is a disease with unclear etiology and unclear pathogenesis of the autoimmune system. Its main pathological change is systemic vasculitis, commonly occurring in the coronary arteries. At the onset, symptoms in the hands and feet include stiff swelling and erythema during the acute phase. In the recovery phase, membranous peeling occurs at the junction of the skin and the nail base of fingers and toes, and there are transverse grooves in the nails. In severe cases, the nails of the fingers and toes may also fall off. Additionally, its skin manifestations include polymorphic skin plaques and scarlet fever-like rashes, which commonly appear in the first week of the disease, with redness and peeling of the skin around the anus.
What is the treatment method for rickets?
Rickets is primarily caused by a deficiency in vitamin D, leading to disturbances in calcium and phosphorus metabolism in the body, which results in abnormal skeletal development—a type of systemic nutritional disease. The most common form is nutritional rickets due to a deficiency in nutrients and vitamin D. Treatment focuses on controlling the active phase to prevent relapse of deformities. Therefore, early detection and comprehensive treatment are crucial. The primary treatment method involves supplementing with vitamin D. The choice of vitamin D, the dosage, and the length of treatment generally need to be tailored to the specific circumstances of the child. Additionally, increased exposure to sunlight and appropriate outdoor activities should be encouraged to enhance the absorption of vitamin D. Children should also be supplemented with calcium and a variety of vitamins. Follow-ups during the recovery phase are necessary, and training in sitting, standing, and walking should not be started too early to avoid deformities in the lower limbs. Furthermore, no medication is needed during the residual stage of the disease. For mild to moderate cases, physical exercise should be intensified, and skeletal deformities can be corrected using active or passive exercises. Severe skeletal deformities may require surgical correction.
Why does rickets cause sweating?
Rickets is a chronic, nutritional disease characterized by skeletal lesions due to a deficiency of vitamin D in the body, leading to disturbances in calcium and phosphorus metabolism. It is primarily caused by a lack of vitamin D, which can increase neural excitability. Research shows that the vitamin D metabolite, 1,25-dihydroxyvitamin D3, is not only an essential nutrient but also a precursor to hormones. It is potentially related to various cells including those in the thyroid, pancreas, stomach, brain, and those involved with calcium balance, as well as the immune, endocrine, reproductive systems, skin, and tumors. A deficiency in vitamin D can increase myocardial excitability and lead to symptoms like excessive sweating.