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Yan Xin Liang
Pediatrics
About me
Graduated from Nanhua University, engaged in pediatric work for over 10 years, and have further studied at Hunan Children's Hospital, Xiangya Hospital, and Beijing Children's Hospital.
Proficient in diseases
Diagnosis and treatment of common diseases in pediatric internal medicine, especially skilled in the diagnosis and treatment of pediatric respiratory system and neurological diseases, such as pediatric convulsions, epilepsy, asthma, and lung infections...
Voices
Can children with diarrhea eat sugar?
For children with diarrhea, it is best to stick to a light, easily digestible diet. During diarrhea, a child's gastrointestinal function may decline, leading to indigestion or gastrointestinal disorders. In such cases, one must consume foods that are easy to digest, avoiding greasy, spicy, stimulative, and hard-to-digest foods. Meanwhile, sugar should also be largely avoided, as many instances of diarrhea in children are caused by lactose intolerance. Adding sugar under these circumstances can exacerbate the symptoms of diarrhea. Furthermore, some cases of diarrhea in children may lead to temporary lactose intolerance. In such situations, sugar should not be added as well. Thus, when dealing with diarrhea, it is advisable to refrain from consuming sugar.
Can phenylketonuria be cured?
Once phenylketonuria is diagnosed, immediate treatment is necessary; the younger the age at which treatment is started, the better the prognosis. The main approach is to use a low phenylalanine formula milk for treatment. Once the blood phenylalanine concentration has decreased to the desired level, we can gradually begin adding small amounts of natural diet. For small infants, breast milk is preferred since it contains much less phenylalanine than cow's milk. For older infants, additions such as cow's milk, porridge, noodles, and eggs are acceptable, but supplements should still follow the principle of being low in protein and phenylalanine. The amount and frequency of these additions should be determined based on the phenylalanine concentration, as too high or too low levels can affect growth and development. This condition can only be managed by controlling symptoms and maintaining blood phenylalanine levels. Long-term treatment is required; it cannot be cured, only controlled.
Early symptoms of childhood leukemia
Generally speaking, leukemia can manifest symptoms related to a decrease in all three blood cell lines. Changes in these three lines typically include an increase or decrease in white blood cells, a decrease in red blood cells leading to symptoms of anemia, and a reduction in platelets, resulting in symptoms of bleeding. Children with leukemia often experience recurrent fevers and are prone to infections, as well as bleeding gums. There may also be enlargement of the liver, spleen, and lymph nodes, along with pallor of the face and lips, which appear pale and bloodless.
How many days does the rash usually subside in young children with sudden onset?
How many days does it take for infantile roseola to fade? Typically, infants with roseola may experience a fever for three to five days, followed by the appearance of the rash, which usually lasts about three days. After the rash fully emerges, it will gradually fade away. Therefore, the rash from infantile roseola might appear as red, rose-like rashes, spreading across various parts of the body. It usually starts to subside slowly after 2 to 3 days, leaving no marks or skin scars. Unlike measles, where the skin can feel rough, pitted, or flaky after the rash subsides, the skin surface after recovering from roseola is very smooth, leaving no traces.
Is hand, foot, and mouth disease contagious?
Hand, foot, and mouth disease is a contagious disease caused by an infection of enteroviruses. There are more than 20 types of enteroviruses that cause the disease, with Coxsackievirus A16 and Enterovirus 71 being the most common. The main clinical manifestations of this disease include blisters on the hands, feet, mouth, and buttocks. Some patients may experience fever, while others may have no fever or only a mild fever. The disease is contagious and primarily transmitted through contact. For example, it can be spread through saliva, droplets, hand-to-mouth contact, and contact with the secretions from the blisters, among other methods.
Phenylketonuria is caused by how?
Phenylketonuria is caused by how? It is an autosomal recessive genetic disease, mainly due to a mutation in the phenylalanine hydroxylase gene, which leads to a reduction in the activity of phenylalanine hydroxylase. Phenylalanine is an essential amino acid in the human body. The phenylalanine ingested is partly used for protein synthesis, and the rest is converted into tyrosine by the action of phenylalanine hydroxylase, with only a small amount of phenylalanine being converted into phenylpyruvic acid through a minor metabolic pathway under the action of transaminase. Due to the reduced activity of phenylalanine hydroxylase, phenylalanine cannot be converted into tyrosine, resulting in an extremely high concentration of phenylalanine in the blood, cerebrospinal fluid, and tissues. Through a bypass metabolism, a large amount of phenylpyruvic acid, phenylacetic acid, phenyllactic acid, and p-hydroxyphenylacetic acid are produced. High concentrations of phenylalanine and its metabolites thus lead to brain damage. Due to the synthesis disorder of tyrosine, this also leads to symptoms such as light skin and hair pigmentation.
Early symptoms of baby's scalp ringworm
Baby tinea capitis is a disease caused by fungal infection. The initial signs are an increase in dandruff and hair that breaks easily. Generally, there are different types based on these symptoms. However, with the gradual improvement of sanitary conditions, the incidence of tinea capitis is now very low and not commonly seen in clinical settings. If tinea capitis occurs, treatment should follow the principles of shaving the head, washing the hair, applying medication, disinfecting, and taking medication. Since fungi thrive in poor sanitary conditions, it is necessary to shave the child's hair and clean and disinfect the scalp, while also taking some antifungal medications. With these measures, the treatment of tinea capitis can achieve good results.
What is the cause of diarrhea in children?
Childhood diarrhea is divided into infectious diarrhea and non-infectious diarrhea, with the common type in children being infectious diarrhea, which includes rotavirus enteritis, bacterial enteritis, and bacterial dysentery, among others, with rotavirus enteritis being the most common. It is characterized by watery diarrhea or "egg-flower" style diarrhea. It is also accompanied by dry mouth, reduced urine output, and some can have symptoms such as vomiting and fever. Similarly, bacterial diarrhea can also present with vomiting, fever, and other symptoms. Other types of diarrhea, such as allergic diarrhea, commonly occur from milk allergies, and there are cases of lactose intolerance that also result in diarrhea. Therefore, there are many causes of childhood diarrhea, and each case should be analyzed specifically.
Clinical manifestations of childhood diarrhea
Pediatric diarrhea is caused by a variety of pathogens and factors, characterized by an increase in the frequency of bowel movements and changes in the characteristics of stools. It is one of the most common diseases in infants and young children in China, with a high incidence in infants and young children from six months to two years old, half of which are under one year old. It is one of the main causes of malnutrition and growth and development disorders in children. Diarrhea has some common manifestations, including loss of appetite, occasional regurgitation or vomiting, increased frequency of bowel movements, watery or thin stools that are yellow or yellow-green, some with a sour smell. In severe cases of diarrhea, it is important to check for dehydration and electrolyte disorders, as well as symptoms of systemic infection and intoxication, such as fever, irritability or lethargy, drowsiness, pale complexion, and even coma and shock.
The pathogenesis of phenylketonuria
Phenylketonuria is an autosomal recessive genetic disorder. Phenylalanine, which is an essential amino acid required by the human body, is partly used for protein synthesis after ingestion and partly converted into tyrosine via the action of phenylalanine hydroxylase, which is necessary for the synthesis of substances such as adrenaline, melanin, and thyroxine. Phenylketonuria is mainly caused by a deficiency of phenylalanine hydroxylase, which prevents the conversion of phenylalanine to tyrosine. This leads to increased levels of phenylalanine in the blood, cerebrospinal fluid, and various tissues. Additionally, as the primary metabolic pathway is blocked, secondary metabolic pathways are enhanced. Under the action of transaminases, phenylalanine undergoes deamination to produce large quantities of phenylpyruvic acid, which is further metabolized to produce phenylacetic acid, phenyllactic acid, and p-hydroxyphenylpyruvic acid, among other byproduct metabolites. These are excreted in large amounts in the urine. The high concentration of phenylalanine and its byproduct metabolites accumulates in brain tissue, leading to damage to brain cells and resulting in a range of clinical symptoms.