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Yan Xin Liang
Pediatrics
About me
Graduated from Nanhua University, engaged in pediatric work for over 10 years, and have further studied at Hunan Children's Hospital, Xiangya Hospital, and Beijing Children's Hospital.
Proficient in diseases
Diagnosis and treatment of common diseases in pediatric internal medicine, especially skilled in the diagnosis and treatment of pediatric respiratory system and neurological diseases, such as pediatric convulsions, epilepsy, asthma, and lung infections...
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Can children with roseola be exposed to wind?
Pediatric emergency care for young children involves dealing with acute viral infections causing feverish illnesses, where the fever can persist for 3 to 5 days. During this period, if going outside, it's important to keep warm to prevent further worsening of cold symptoms. Therefore, appropriate protective measures should be taken when going out. As for exposure to the wind, a slight breeze isn't usually a major issue, especially after the body temperature has peaked - a bit of wind is generally harmless. However, during the rising phase of body temperature when the hands and feet are cold, exposure to wind can cause the baby to experience further symptoms of cold aversion, which is detrimental to the baby. Therefore, it is still important to take protective measures and avoid wind exposure as much as possible.
What can children eat when they have diarrhea?
Childhood diarrhea is a manifestation of acute gastroenteritis. During this process, his digestive function will definitely be affected, so it is important to pay attention to diet. Provide light and easy-to-digest foods. If breastfeeding, the mother's diet should also include light and easy-to-digest foods. For babies fed with formula, we can use diarrhea-specific formula, and supplementary foods such as rice porridge and noodles can be added. Avoid greasy, spicy, and irritating foods. It is appropriate to add vegetables, but do not orally administer sugary foods, including sugars, biscuits, etc., as these are not suitable for the baby.
How long does Kawasaki disease require hospitalization?
Kawasaki disease is an autoimmune vasculitis, primarily concerning because it affects the coronary arteries of the heart. Without effective treatment, some cases can lead to coronary artery dilation and even the formation of coronary artery aneurysms. Therefore, hospitalization is generally recommended during the acute phase of Kawasaki disease. During hospitalization, treatment typically involves the use of intravenous immunoglobulin and aspirin. After treatment with intravenous immunoglobulin, the body temperature generally begins to decrease gradually. Once the body temperature has stabilized for about three days and there are no complications, the patient can usually be discharged, so most hospital stays last about 5-7 days. (Medication should be taken under the guidance of a doctor.)
How to deal with baby roseola
For the nursing methods of infantile sudden rash, it is important to note that first, the child should drink more water to replenish sufficient fluids. The initial symptom of infantile sudden rash is fever, but too high a body temperature can cause excessive loss of water in the baby, and drinking more water not only replenishes fluids but also helps to reduce fever, Secondly, physical cooling should be used. During periods of high fever, physical cooling methods such as using cold packs or warm water baths should be employed to help reduce the child's body temperature while maintaining cleanliness and hygiene, During the child's visit to the clinic, it is necessary to keep the indoor environment ventilated and clean, bathe the baby in a timely manner, and keeping the skin fresh is also beneficial for eliminating toxins. Additionally, a healthy diet is crucial during the emergency period; although the baby might not show abnormalities, attention must be paid to the diet, especially for breastfed babies, where the mother herself must ensure a healthy and nutritious diet.
Why does infantile eczema cause asthma?
There are many causes of infantile eczema, mainly indicating that the baby is experiencing allergic phenomena. These allergies can be due to internal factors or exposure to certain allergens or food allergies, all leading to eczema. Often, children with this type of eczema have an allergic constitution, making them prone to developing asthma. Therefore, when babies with asthma come for a consultation, it is routine to ask if they have a history of eczema, as having eczema often suggests an allergic constitution. It's not that infantile eczema causes asthma, but rather, babies with this constitution are prone to asthma attacks.
If phenylketonuria is not treated, what will happen?
Phenylketonuria (PKU) is an amino acid metabolic disorder primarily caused by a deficiency in phenylalanine hydroxylase. This deficiency prevents phenylalanine from being converted to tyrosine, leading to a significant accumulation of phenylalanine, which is then excreted in large quantities in the urine. It is a common genetic metabolic disorder. If not treated, it can lead to developmental delays in children, particularly in intellectual development, with markedly lower IQ than peers, especially noticeable in language development disorders. Additionally, it can also present with neurological and psychiatric symptoms such as agitation, hyperactivity, increased muscle tone, and hyperreflexia. There is also depigmentation of the skin and hair, and the urine may have a mousy odor.
Phenylketonuria patients lack phenylalanine hydroxylase.
Phenylketonuria is an autosomal recessive genetic disorder caused by mutations in the phenylalanine hydroxylase gene, leading to reduced enzyme activity and resulting in the accumulation of phenylalanine and its metabolic products in the body, causing the disease. Phenylketonuria is the most common congenital amino acid metabolism disorder, clinically presenting with intellectual developmental delays, lighter skin and hair pigmentation, and a musty urine odor. The disease is mainly caused by mutations in the phenylalanine hydroxylase gene, leading to reduced enzyme activity.
What should a child with pneumonia eat?
Childhood pneumonia is an infectious disease that not only presents symptoms related to the respiratory system but also may include symptoms from the gastrointestinal tract, such as vomiting, diarrhea, loss of appetite, and abdominal distension. Therefore, the diet for pneumonia must be light and easy to digest. For instance, if the child is breastfed, the mother should consume a light and digestible diet. If the child is fed with cow's milk, the milk can be diluted appropriately to aid digestion. Supplementary foods can generally include thin porridge, noodles, rice paste, vegetables, and fruits, as well as a moderate amount of lean meat and eggs, but not in excess. Oily and spicy foods should not be included.
What can be eaten with phenylketonuria?
Phenylketonuria is primarily due to a deficiency of phenylalanine hydroxylase in the body, which prevents the conversion of phenylalanine to tyrosine. Due to the blockage of the metabolic pathway, secondary metabolic pathways are enhanced, leading to the deamination of phenylalanine and the production of large amounts of phenylpyruvic acid. Through oxidation, by-products such as phenylacetic acid, phenyllactic acid, and para-hydroxyphenylpyruvic acid are formed. The treatment mainly involves the use of low-phenylalanine formula milk. When the concentration in the blood reaches the desired level, natural foods can be gradually added in small amounts. Breast milk is the preferred choice as it contains only one-third of the phenylalanine content of cow's milk. For older infants and children, cow's milk, porridge, noodles, and eggs can be added. The principle for introducing foods should be based on low protein and low phenylalanine content, adjusted according to the phenylalanine concentration in the blood. Both too high and too low levels of phenylalanine can affect growth and development.
Is phenylketonuria characterized only by pale facial skin?
Phenylketonuria is a common amino acid metabolic disorder. It is primarily due to the deficiency of phenylalanine hydroxylase, which prevents the conversion of phenylalanine to tyrosine, leading to a series of clinical symptoms. These can manifest as intellectual disability, neuropsychiatric symptoms, eczema, skin scratching, pigment loss, and a musty odor. The skin whiteness mentioned is not merely facial pallor; it affects the entire skin because the benefits of tyrosine are reduced, leading to decreased melanin synthesis. Consequently, the hair of affected children can also be lighter and tend to be brown.