Does patent ductus arteriosus cause easy sweating?

Written by Di Zhi Yong

Cardiology

Updated on September 07, 2024

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The arterial duct is prone to sweating. During this period, it is best to advise patients to monitor changes in blood pressure, heart rate, and pulse. Sometimes there may be incidences of rapid heart rate, as this is a type of congenital heart disease. If a patient is prone to respiratory infections, especially pneumonia, this condition can lead to a decrease in resistance. For such patients with weak constitution, they are more likely to sweat. In terms of treatment, early surgical intervention is advised to alleviate the sweating, and preparations should be made early on.

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Written by Xie Zhi Hong

Cardiology

1min 37sec

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Why does patent ductus arteriosus cause differential cyanosis?

The arterial duct refers to a blood vessel connecting the main pulmonary artery and the aorta, which is normally present during fetal development. It allows blood from the mother's placenta to seep into the pulmonary artery and then enter the aorta to supply the entire body's vasculature. After the birth of an infant, this blood vessel naturally closes, typically within 3 months, and most people will have it closed by 1 year. If it has not closed after 1 year, it is referred to as patent ductus arteriosus. Because the arterial duct can connect the aorta and the main pulmonary artery, blood from the systemic circulation in the aorta may directly return to the pulmonary artery at this time. If the pulmonary artery blood flow significantly increases, it can lead to pulmonary arterial hypertension and might also cause right ventricular hypertrophy. Increased cardiac workload can lead to right-sided heart failure and may also cause left-sided heart failure. When severe left-sided heart failure occurs, a right-to-left shunt can develop, which allows pulmonary artery blood to directly enter the aorta, causing venous blood to be delivered directly into the systemic circulation. At this point, differential cyanosis occurs, characterized by bluish discoloration of the upper limbs and mild blueness in the lower limbs.

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Written by Cai Li E

Cardiology

55sec

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Can you run with a patent ductus arteriosus?

Patients with patent ductus arteriosus should avoid running. The condition can cause a left-to-right shunt, increasing pulmonary circulation and the volume of blood returning to the left heart system, which in turn leads to increased left heart workload and potential left heart dysfunction. In adults with patent ductus arteriosus, the clinical symptoms vary depending on the size of the shunt. Those with very small shunts may exhibit no subjective symptoms clinically. Patients with moderate shunts often experience symptoms such as fatigue, palpitations after activity, shortness of breath, and chest tightness. Those with large shunts, often accompanied by severe secondary pulmonary hypertension, may experience a right-to-left shunt, leading to cyanosis and very severe clinical symptoms.

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Written by Chen Tian Hua

Cardiology

49sec

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Will patent ductus arteriosus cause mitral regurgitation?

Patent ductus arteriosus usually does not cause mitral regurgitation. Since the pressure in the aorta is greater than that in the pulmonary artery, blood from the aorta will shunt to the pulmonary artery, significantly increasing blood flow in the pulmonary circulation. This eventually leads to pulmonary hypertension, causing an increased afterload on the right ventricle. Over time, an increased afterload on the right ventricle can lead to right ventricular enlargement and right heart failure, resulting in dilation of the tricuspid annulus and tricuspid regurgitation due to incomplete closure. Patients with patent ductus arteriosus need to seek prompt medical assessment to determine the need for surgical treatment. For patients with surgical indications, surgery should be administered promptly to avoid the adverse consequences of long-term blood shunting.

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Written by Xie Zhi Hong

Cardiology

39sec

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Can a patent ductus arteriosus be treated with minimally invasive surgery?

Patent ductus arteriosus refers to the condition in infants where the ductal connection between the pulmonary artery and the aortic arch from the embryonic period remains open after the first year of birth. Generally, this duct is tubular or funnel-shaped and most cases can be treated minimally invasively. Surgical treatment is considered only if there are severe adhesions involving the aorta or pulmonary artery, or there are developmental issues. Therefore, patent ductus arteriosus can be treated with minimally invasive surgery, and most cases are amenable to such treatment.

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Written by Xie Zhi Hong

Cardiology

1min 4sec

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Can patent ductus arteriosus be treated with medication?

The ductus arteriosus is a blood vessel connecting the pulmonary artery to the aorta in the fetal heart, primarily used during the embryonic stage to supply blood from the mother to the heart, thus providing sufficient oxygenated blood for fetal development and growth. Generally, the ductus arteriosus should close automatically within three months after birth. If it remains open for over a year, it may indicate a congenital heart defect known as patent ductus arteriosus. Previously, high doses of aspirin were used to treat this condition, but this treatment was not very effective. Patients with patent ductus arteriosus should instead consider minimally invasive interventional treatments to seal the duct, or surgical ligation can be performed. However, surgical ligation may cause significant trauma or scarring to the patient. The majority of cases with patent ductus arteriosus can be treated using minimally invasive methods.