Can people with cerebral infarction drink alcohol?

Written by Gao Yi Shen
Neurosurgery
Updated on January 03, 2025
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Patients with cerebral infarction should not drink alcohol. Although alcohol can have certain effects in promoting blood circulation and removing blood stasis, the specific content of alcohol and its degree, as well as each person's tolerance, vary. While in some cases, certain components can promote blood circulation, this does not mean it always activates blood circulation. In fact, excessive promotion of blood circulation at times can even lead to cerebral hemorrhage and other changes in medical conditions, posing even greater risks to patients. Another point is that drinking alcohol can cause excitement, which may lead to increased blood pressure. If there are plaques or other impurities in the arteries, they might dislodge and lead to further exacerbation of cerebral infarction. Therefore, it is also advised not to drink alcohol for these reasons.

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Written by Shu Zhi Qiang
Neurosurgery
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How to rehabilitate from a cerebral infarction?

Brain infarction is one of the most common cerebrovascular accidents, accounting for about 65% of all cerebrovascular diseases. The types of brain infarction include lacunar infarction, cerebral thrombosis, and cerebral embolism; sometimes it also includes cerebral venous sinus thrombosis. Any type of brain infarction, or cerebral infarction, can cause certain neurological dysfunctions. Therefore, once the patient's condition is stabilized, it is advisable to actively consider rehabilitation treatments. These rehabilitation treatments mainly include hyperbaric oxygen therapy, acupuncture rehabilitation, and physical rehabilitation therapy.

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Written by Chen Yu Fei
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Symptoms of cerebral infarction

For patients with cerebral infarction, during the acute phase, they typically exhibit clear symptoms of headache, dizziness, nausea, and vomiting. Patients may experience slurred speech, difficulty speaking, and obvious articulation disorders. Additionally, some patients may develop facial nerve paralysis, deviation of the mouth to one side, drooling, and often exhibit clinical signs such as a shallower nasolabial fold and disappearance of forehead wrinkles. Furthermore, some patients may also experience dysphagia, manifested by coughing when drinking water, and may be accompanied by aphasia or motor dysfunction, primarily presenting as motor aphasia, mixed aphasia, anomic aphasia, and resulting unilateral limb paralysis.

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Written by Hao Li Bin
Neurosurgery
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How is a cerebral infarction treated?

The treatment principles for cerebral infarction include thrombolysis, anticoagulation, volume expansion, lowering blood pressure, and brain protection. The main goal is to improve the blood circulation in the ischemic area of the brain as soon as possible and promote the recovery of neurological functions. During the acute phase, patients should rest in bed as much as possible, enhance care for skin, oral cavity, respiratory tract, and excretion, and maintain electrolyte balance. If a patient still cannot eat 48 to 72 hours after onset, nasogastric feeding with liquid nutrition should be provided to ensure nutritional supply. The priority should be given to the patient's daily care, diet, and the management of other comorbidities. Since some patients with cerebral infarction cannot take care of themselves during the acute phase and may even have difficulty swallowing, without adequate nutrition, metabolic issues can arise quickly. In such cases, even the best medications can fail to achieve positive outcomes. Since cerebral thrombosis is the most common type of cerebral infarction characterized by high incidence, high mortality, high recurrence rate, and high disability rate, patients may experience mild hemiplegia or severe scenarios leading to loss of life. Therefore, lifelong medication is necessary to prevent recurrence, achieve secondary prevention, and ultimately reduce the recurrence of cerebral infarction.

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Written by Gao Yi Shen
Neurosurgery
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Is a cerebral infarction serious?

Stroke is a very serious disease; its essence lies in the ischemic necrosis of nerve cells. A characteristic of nerve cells is that once they undergo ischemic necrosis, they cannot regenerate. Along with cardiac muscle cells, they are the only cells in the human body that cannot regenerate once they have died. Therefore, for the part of the tissue where nerve cells have already died, such as in cases of cerebral thrombosis or stroke where the tissue has already been affected, it is impossible to completely restore it. If the affected area is relatively small, some compensation might be possible. However, if the affected area is relatively large, this will lead to a significant amount of ischemic necrosis in the neural tissue, which can cause total dysfunction of various tissue structures in the body, and in more severe cases, can lead to coma and eventually the death of the patient. Therefore, it is imperative to take this seriously in clinical practice.

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Written by Gao Yi Shen
Neurosurgery
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Premonitory symptoms of cerebral infarction recurrence

The symptoms preceding the recurrence of a cerebral infarction mainly involve observing the symptoms presented during the initial incidence of the stroke. In many cases, the recurrent symptoms are very similar to those initially experienced. For example, if dizziness was a symptom during the previous episode, then dizziness could also be a precursor symptom in a recurrence, with similar nature and intensity of dizziness as before. Additionally, if previous symptoms included limb numbness, weakness, or speech impairment, similar signs might precede a recurrence. Clinically, it is imperative to address these issues before an actual recurrence occurs, such as actively pursuing antiplatelet aggregation therapy, lipid-lowering, and stabilizing plaques, and resorting to surgical treatment if necessary, to better reduce the chances of stroke recurrence.