What location is used for moxibustion in the treatment of cerebral infarction?

Written by Chen Yu Fei
Neurosurgery
Updated on September 28, 2024
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For patients with cerebral infarction, it is usually recommended during the acute phase to conduct a cranial MRI to determine the location and severity of the stroke and to decide the next treatment plan. If surgery is indicated, treatment can be administered through intravenous thrombolysis. If surgery is not indicated, or the optimal treatment time has been missed, it is recommended that the patient be hospitalized for treatment. During the acute phase, medications that invigorate the brain and improve blood circulation while nourishing the nerves are administered, and it is important to monitor any changes in the patient's condition. The effectiveness of moxibustion in treating cerebral infarction is generally moderate. During the recovery phase, patients can try moxibustion, but it is generally advised to seek treatment under the guidance of a Traditional Chinese Medicine physician at a reputable tertiary hospital.

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Written by Gao Yi Shen
Neurosurgery
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Is a cerebral infarction serious?

Stroke is a very serious disease; its essence lies in the ischemic necrosis of nerve cells. A characteristic of nerve cells is that once they undergo ischemic necrosis, they cannot regenerate. Along with cardiac muscle cells, they are the only cells in the human body that cannot regenerate once they have died. Therefore, for the part of the tissue where nerve cells have already died, such as in cases of cerebral thrombosis or stroke where the tissue has already been affected, it is impossible to completely restore it. If the affected area is relatively small, some compensation might be possible. However, if the affected area is relatively large, this will lead to a significant amount of ischemic necrosis in the neural tissue, which can cause total dysfunction of various tissue structures in the body, and in more severe cases, can lead to coma and eventually the death of the patient. Therefore, it is imperative to take this seriously in clinical practice.

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Written by Chen Yu Fei
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Precursors to cerebral infarction

For patients with cerebral infarction, before the onset of the illness, they often lack specific prodromal symptoms. Some patients may experience mild headaches, dizziness, nausea, and a clear feeling of discomfort. Additionally, some patients might experience numbness in the legs and involuntary twitching of facial muscles before the onset. Subsequently, some patients may also start to have drooping of the corner of the mouth to one side, drooling, slurred speech, and distinct articulation disorders. When these clinical presentations occur, it is necessary to be vigilant and seek timely medical attention at a local hospital. A cranial CT or MRI should be performed to confirm the diagnosis.

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Stroke treatment methods

For patients who suffer a stroke, it is crucial to immediately transport them to the nearest local hospital. At the emergency room of the local hospital, a cranial CT scan or MRI should be conducted to confirm the presence of a stroke, as well as to determine its exact range and severity. If there is an acute indication for surgery, thrombolytic therapy is recommended. If the optimal time window for thrombolysis has passed, it is advisable to use medications that invigorate the brain, improve blood flow to remove stasis, nourish the nerves, and enhance microcirculation in the brain tissue. This can effectively help improve the conditions of ischemia and hypoxia in the brain nerves. Furthermore, further treatment should be adjusted based on the patient’s condition. During treatment, attention should be paid to the prevention and treatment of various complications or comorbidities, such as pulmonary infections, urinary tract infections, hypoproteinemia, deep vein thrombosis, etc.

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Written by Shu Zhi Qiang
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post-stroke sequelae

Brain infarction is a very common type of ischemic cerebrovascular disease, which mainly includes the following types: the most common are lacunar infarction, cerebral thrombosis, and cerebral embolism. The neurological sequelae of lacunar infarction are not particularly typical, and patients may exhibit mild incomplete paralysis of limbs, decreased muscle strength, reduced sensation, or ataxia. Cerebral thrombosis and cerebral embolism often cause extensive brain infarction, where patients typically experience severe limb paralysis, loss of sensation, increased tendon reflexes, positive pathological reflexes, and an inability to care for themselves.

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Causes of Cerebral Infarction

The specific causes of cerebral infarction are mostly seen in older patients with several underlying diseases, such as hypertension, diabetes, and hyperlipidemia. Over time, without well-controlled blood pressure, blood sugar, and blood lipids, arteriosclerosis can easily form within the blood vessels, or atherosclerotic plaques can develop. Once the plaque forms, under certain triggers, the unstable detachment of the vascular plaque can occur. The embolus then moves with the blood flow into the brain and lodges in a narrowed part of the vessel, causing local blood supply areas to experience narrowing or blockage. At this point, a cerebral infarction is likely to form, leading to ischemia, necrosis, and degeneration of the brain tissue supplied by the vessel, resulting in a softening lesion. On a cranial CT scan and MRI, a local low-density shadow can be observed, which is the specific cause of the cerebral infarction.