Can pulmonary embolism cause bloating and abdominal distension?

Written by An Yong Peng
Pulmonology
Updated on January 04, 2025
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Pulmonary embolism can, in rare cases, cause bloating and abdominal distension. The primary clinical manifestations of pulmonary embolism are chest tightness and difficulty breathing. Severe pulmonary embolism can also lead to a drop in blood pressure and sometimes may even cause the patient to faint. However, it is important to note that in severe cases of pulmonary embolism, patients may also experience abdominal distension. Patients with pulmonary embolism might also have acute pulmonary heart disease, which could include symptoms of gastrointestinal congestion. In such cases, patients may experience abdominal bloating. Additionally, there is a special condition known as chronic thromboembolic pulmonary hypertension, where patients may experience chronic pulmonary heart disease and chronic hypoxia, which can also lead to abdominal distension.

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Written by Wei Shi Liang
Intensive Care Unit
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Can pulmonary embolism lead to pneumonia?

Pulmonary embolism itself does not cause pneumonia; however, during the treatment of pulmonary embolism, procedures such as oral tracheal intubation and the creation of an artificial airway might be employed. These can lead to nosocomial infections of iatrogenic origin, resulting in pneumonia. Pulmonary embolism often manifests as unexplained respiratory difficulty, pleuritic pain, tachycardia, and decreased oxygen saturation. Other high-risk factors include being over the age of 40, having a history of DVT (Deep Vein Thrombosis), obesity, prolonged immobilization, stroke, congestive heart failure, malignancy, lower limb fracture, anesthesia time exceeding 30 minutes during surgery, pregnancy and childbirth, use of estrogen, and a hypercoagulable state. These are all potential high-risk factors for pulmonary embolism.

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Written by Wang Li Bing
Intensive Care Medicine Department
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What are the precursors of pulmonary embolism?

Pulmonary embolism is also relatively common in clinical practice. Its precursors may include varying degrees of respiratory difficulty, chest tightness, and shortness of breath, among others. If such symptoms occur, medical attention should be sought promptly, primarily to rule out the possibility of acute myocardial infarction. Pulmonary artery CTA can further confirm whether there is a pulmonary embolism. In cases of extensive pulmonary embolism, there is a high risk of sudden death, and aggressive thrombolytic and anticoagulation treatments should be administered. If the patient's chest tightness and shortness of breath are relieved after the aforementioned treatments, hospitalization for observation and treatment is still necessary.

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Written by Wei Shi Liang
Intensive Care Unit
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Is pulmonary embolism related to pneumonia?

If unexplained shortness of breath, pleuritic chest pain, tachycardia, and decreased oxygen saturation occur, one should be highly vigilant about pulmonary embolism. Generally, pulmonary embolism has no direct relationship with pneumonia. High-risk factors for pulmonary embolism include obesity, prolonged immobilization, stroke, congestive heart failure, malignant tumor, inflammatory bowel disease, lower limb fracture, anesthesia time exceeding 30 minutes, and acquired or genetic hypercoagulable state. It usually manifests as difficulty breathing, rapid breathing, and pleuritic chest pain.

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Written by Wang Chun Mei
Pulmonology
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Will acute pulmonary embolism get better in two days?

Acute pulmonary embolism is a type of disease with very many and complex inducing factors. Clinically, acute pulmonary embolism has a very sudden onset, and patients often present with unexplained symptoms such as pale complexion, chest tightness, chest pain, and difficulty breathing. During the acute phase of pulmonary embolism, immediate and effective anti-shock and other symptomatic resuscitation are essential upon arrival at the hospital. Generally, after timely and effective resuscitation, the patient's condition may be somewhat controlled. Usually, the first 1-3 days after the onset of pulmonary embolism are the most dangerous; therefore, continuous monitoring of the patient's vital signs is crucial during this time, followed by anti-shock and anticoagulation treatment as needed. Therefore, acute pulmonary embolism will not improve by the second day, although some of the patient's clinical symptoms may slightly improve after treatment.

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Written by Wei Shi Liang
Intensive Care Unit
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Pulmonary Embolism Interventional Treatment Methods

Interventional treatment for pulmonary embolism is mainly used for large pulmonary embolisms in the main trunk or major branches of the pulmonary artery. It is applicable in the following scenarios: contraindications to thrombolysis and anticoagulation therapy, inefficacy after thrombolysis or aggressive medical treatment, or lack of surgical conditions. Interventional therapy for pulmonary embolism can involve removing the embolus or breaking it into fragments, allowing it to move to the distal pulmonary arteries, thereby opening the central pulmonary arteries, rapidly reducing pulmonary artery resistance, significantly increasing total pulmonary blood flow, improving cardiopulmonary hemodynamics, and right ventricular function. The treatment involves catheter fragmentation and suction of large clots in the pulmonary artery or performing balloon angioplasty, and it also enables local administration of small-dose thrombolysis. These are the primary methods of interventional treatment for pulmonary embolism.