What tea to drink for arteriosclerosis?

Written by Zhang Yue Mei
Cardiology
Updated on September 09, 2024
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Patients with arteriosclerosis can drink various types of tea, including green tea, black tea, chrysanthemum tea, goji berry tea, and ginseng tea. These teas can be consumed to increase blood flow, renew knowledge, enhance circulation, cleanse the blood vessels, and alleviate arteriosclerosis. However, one should not rely solely on drinking tea to treat arteriosclerosis. It is also necessary to use certain medications and adjust dietary structures, adopting a low-fat, low-salt, low-sugar diet, avoiding animal liver and fatty meats, reducing the intake of fried foods, and abstaining from high-cholesterol foods such as cream, fish roe, and shrimp.

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The difference between arteriosclerosis and atherosclerosis

Arteriosclerosis is a common and important type among a group of vascular diseases known as arteriosclerosis. The common characteristics of various arterioscleroses include thickening and hardening of arterial walls, loss of elasticity, and narrowing of the lumen. Atherosclerosis is a type of arteriosclerosis characterized by lesions starting from the arterial intima, subsequently involving the accumulation of lipids and complex carbohydrates, bleeding, thrombosis, proliferation of fibrous tissues, and deposition of calcium, along with gradual degeneration and calcification of the arterial media. Since the lipids accumulated in the arterial intima appear yellowish and mushy, it is termed atherosclerosis.

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Causes of Arteriosclerosis

Arteriosclerosis, the most common and important type of arteriosclerosis, is characterized by thickening and hardening of the arterial walls, loss of elasticity, and narrowing of the lumen. The causes are currently considered to be related to several factors, including age and gender, with older males being more prone. Patients with abnormal blood lipids, hypertension, smokers, those with diabetes or impaired glucose tolerance, obese patients, and those with a family history are all more likely to develop arteriosclerosis. Due to the incomplete certainty of the causes, its prevention and control are also managed through multiple factors.

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Written by Zeng Wei Jie
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How is arteriosclerosis treated?

To treat arteriosclerosis, we differentiate treatments based on the severity of the condition. For early-stage patients, we emphasize the control of lipids, blood pressure, and blood sugar to potentially slow the progression of arteriosclerosis. For patients who have already suffered damage to target organs, such as cerebral infarction, myocardial infarction, renal artery stenosis, or mesenteric artery stenosis, treatment might significantly rely on medication, and could possibly require the use of stents or even surgical bypass procedures. For the general population, we emphasize primary prevention, which includes a reasonable diet, appropriate exercise, and a balanced mental state to effectively prevent the disease.

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Written by Tang Li
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The difference between arteriosclerosis and atherosclerosis

Arteriosclerosis is the most common and most important among a group of vascular diseases called arteriosclerosis. The common characteristics of various arteriosclerosis include thickening, hardening, loss of elasticity, and narrowing of the arterial walls. The characteristic of arteriosclerosis is that the affected arterial lesions start from the intima, with various lesions coexisting, including local accumulation of papyraceous and complex carbohydrates, proliferation of fibrous tissue, and formation of plaques due to calcification, along with gradual degradation of the arterial media. Secondary lesions include intraplaque hemorrhage, plaque rupture, and local thrombosis formation. Modern cellular and molecular biology techniques show that arteriosclerotic lesions are characterized by migration of macrophages, proliferation of smooth muscle cells, and abundant formation of fibrous, collagen, elastic fibers, and proteoglycans as connective tissue matrices, as well as intra- and extracellular lipid accumulation. Because the lipid accumulation in the intima appears yellow and mushy, it is called arteriosclerosis. Although arteriosclerosis is only one type of arteriosclerosis, it is commonly referred to simply as arteriosclerosis due to its frequent occurrence and significant clinical relevance.

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Causes of arteriosclerosis

The etiology of arteriosclerosis has not been fully determined, and studies indicate that arteriosclerosis is a multifactorial disease, caused by multiple factors acting at different stages, which are referred to as risk factors. The primary risk factors include the following: First, age and gender. Clinically, it is more common in middle-aged and elderly people over forty years old. After the age of forty-nine, the progression is fast, but early arteriosclerotic changes have also been found in autopsy of some young adults and even children. In recent years, clinical onset age tends to be younger. Compared to men, the incidence rate in women is lower, because estrogen has a protective effect against arteriosclerosis. Therefore, the incidence rate in women increases rapidly after menopause. Age and gender are unchangeable risk factors. Second, abnormal lipid levels, with abnormal lipid metabolism being the most important risk factor for arteriosclerosis. Third, hypertension, as the incidence of arteriosclerosis in patients with hypertension is significantly higher. Sixty to seventy percent of patients with coronary arteriosclerosis have hypertension, and patients with hypertension are three to four times more likely to have arteriosclerosis compared to those with normal blood pressure. Fourth, smoking, as the incidence and mortality rate of coronary arteriosclerosis in smokers are two to six times higher than in non-smokers, and it correlates positively with the number of cigarettes smoked daily. Secondhand smoke is also a risk factor. Fifth, diabetes and glucose intolerance, where not only is the incidence of arteriosclerosis in diabetic patients several times higher than in non-diabetics, but the progression of the disease is also rapid. Sixth, obesity, defined as being more than twenty percent over the standard weight or a BMI greater than twenty-four. Obesity is also a risk factor for arteriosclerosis. Seventh, family history, where a family history of coronary heart disease, diabetes, hypertension, and hyperlipidemia significantly increases the incidence of coronary heart disease. Various theories have been proposed to explain the pathogenesis of coronary arteriosclerosis from different perspectives. These include the lipid infiltration theory, thrombosis theory, and smooth muscle cell clonal theory. In recent years, the endothelial damage response theory has gained more support, suggesting that the disease results from various risk factors ultimately damaging the arterial intima, and the formation of arteriosclerosis lesions is an inflammatory, fibro-proliferative response of the arteries to endothelial damage.