How long can one live with arteriosclerosis?

Written by Chen Ya

Geriatrics

Updated on September 13, 2024

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Arteriosclerosis has a certain impact on survival and longevity, but it is not an absolute determinant. People with mild arteriosclerosis have fewer sudden incidents of cerebral infarction and myocardial infarction, thus they may have a longer survival period. However, if arteriosclerosis leads to myocardial infarction or cerebral infarction, it will also affect their lifespan, but there is no absolute conclusion.

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Written by Li Hai Wen

Cardiology

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How to check for arteriosclerosis?

Arteriosclerosis is a relatively common disease in our daily life. After being diagnosed with arteriosclerosis, the following tests should be considered: Firstly, blood tests, such as lipid panels, are necessary to determine the levels of blood lipids. Based on the results, medication might be required for treatment. Secondly, arterial Doppler ultrasound is useful as it can effectively detect the presence and severity of arteriosclerosis. Thirdly, arterial angiography is recommended when there is a strong suspicion of arterial narrowing due to arteriosclerosis, leading to coronary heart disease or other conditions. This test helps in determining the severity of the arteriosclerosis and in deciding the treatment plan.

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Written by Zeng Wei Jie

Cardiology

1min 3sec

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What medicine to take for arteriosclerosis?

Drug therapy is an important component of the treatment for atherosclerosis and mainly includes the following types. The first is to regulate blood sugar and lipids, keeping them under control to slow down the process of atherosclerosis. The second involves taking antiplatelet medications to prevent the formation of blood clots, especially since some plaques, particularly soft plaques, are prone to rupture. For some patients who have already formed blood clots, thrombolytic therapy might be considered, but this is not typically necessary for all patients. Additionally, because high blood pressure can accelerate the progression of atherosclerosis, some patients also need to take antihypertensive drugs. For patients with narrowed blood vessels and severe symptoms, such as angina, vasodilators, including nitrates, might be used. Therefore, the treatment choices may vary depending on the severity of the condition, and the modes of drug therapy differ. (Note: Specific medications should be taken under the guidance of a doctor.)

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Written by Chen Ya

Geriatrics

51sec

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The difference between arteriosclerosis and atherosclerosis

Arteriosclerosis is a common and important type among a group of vascular diseases known as arteriosclerosis. The common characteristics of various arterioscleroses include thickening and hardening of arterial walls, loss of elasticity, and narrowing of the lumen. Atherosclerosis is a type of arteriosclerosis characterized by lesions starting from the arterial intima, subsequently involving the accumulation of lipids and complex carbohydrates, bleeding, thrombosis, proliferation of fibrous tissues, and deposition of calcium, along with gradual degeneration and calcification of the arterial media. Since the lipids accumulated in the arterial intima appear yellowish and mushy, it is termed atherosclerosis.

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Written by Li Hai Wen

Cardiology

39sec

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Can atherosclerosis be cured?

Atherosclerosis is a very common disease in daily life. Many patients, after being diagnosed with atherosclerosis, often ask doctors "Can my atherosclerosis be cured?" The answer is no, as atherosclerosis cannot be completely cured. However, this does not mean that atherosclerosis is untreatable. From a medical perspective, controlling blood pressure, lipids, and blood sugar, maintaining good lifestyle habits, exercising regularly, quitting smoking, controlling weight, and following a doctor's guidance to properly take medications can effectively control the onset and progression of atherosclerosis.

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Written by Tang Li

Cardiology

3min 21sec

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Causes of arteriosclerosis

The etiology of arteriosclerosis has not been fully determined, and studies indicate that arteriosclerosis is a multifactorial disease, caused by multiple factors acting at different stages, which are referred to as risk factors. The primary risk factors include the following: First, age and gender. Clinically, it is more common in middle-aged and elderly people over forty years old. After the age of forty-nine, the progression is fast, but early arteriosclerotic changes have also been found in autopsy of some young adults and even children. In recent years, clinical onset age tends to be younger. Compared to men, the incidence rate in women is lower, because estrogen has a protective effect against arteriosclerosis. Therefore, the incidence rate in women increases rapidly after menopause. Age and gender are unchangeable risk factors. Second, abnormal lipid levels, with abnormal lipid metabolism being the most important risk factor for arteriosclerosis. Third, hypertension, as the incidence of arteriosclerosis in patients with hypertension is significantly higher. Sixty to seventy percent of patients with coronary arteriosclerosis have hypertension, and patients with hypertension are three to four times more likely to have arteriosclerosis compared to those with normal blood pressure. Fourth, smoking, as the incidence and mortality rate of coronary arteriosclerosis in smokers are two to six times higher than in non-smokers, and it correlates positively with the number of cigarettes smoked daily. Secondhand smoke is also a risk factor. Fifth, diabetes and glucose intolerance, where not only is the incidence of arteriosclerosis in diabetic patients several times higher than in non-diabetics, but the progression of the disease is also rapid. Sixth, obesity, defined as being more than twenty percent over the standard weight or a BMI greater than twenty-four. Obesity is also a risk factor for arteriosclerosis. Seventh, family history, where a family history of coronary heart disease, diabetes, hypertension, and hyperlipidemia significantly increases the incidence of coronary heart disease. Various theories have been proposed to explain the pathogenesis of coronary arteriosclerosis from different perspectives. These include the lipid infiltration theory, thrombosis theory, and smooth muscle cell clonal theory. In recent years, the endothelial damage response theory has gained more support, suggesting that the disease results from various risk factors ultimately damaging the arterial intima, and the formation of arteriosclerosis lesions is an inflammatory, fibro-proliferative response of the arteries to endothelial damage.