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21
Chen Guang Yin
Cardiology
About me
Chief physician, medical doctor, master's supervisor, graduated from the China Academy of Traditional Chinese Medicine, engaged in clinical work for over thirty years.
Proficient in diseases
Specializes in the integrated treatment of cardiovascular diseases (such as hypertension, hyperlipidemia, coronary heart disease, arrhythmia, myocarditis, etc.) and blood system diseases (anemia, leukopenia, thrombocytopenia, etc.) with both Chinese and Western medicine.
Voices
How to self-rescue from sudden cardiac death
Sudden cardiac death is now also a very critical illness often occurring in coronary heart disease. We frequently see news reports of sudden deaths at airports and public places. At such times, self-rescue is extremely difficult. Unless you have a history of coronary heart disease and carry medications with you, if you experience symptoms like chest tightness and shortness of breath, you could take these medications to help yourself. Otherwise, sudden cardiac death occurs abruptly, causing the heart to stop beating within a very short period, and the patient cannot self-rescue.
Is myocardial infarction class II serious?
New York Heart Association (NYHA) Class II heart function is not severe. Myocardial infarction is a relatively serious type of coronary heart disease, which occurs due to the blockage of the coronary arteries leading to myocardial necrosis, and it carries certain risks. It can ultimately lead to complications such as sudden death and heart failure. At this stage, with Class II heart function, symptoms of heart failure can appear under conditions of significant exertion, including chest tightness, breathlessness, fatigue, and shortness of breath. These symptoms correspond to a relatively less severe stage of heart failure.
Is it normal after the complete repair of Tetralogy of Fallot?
Tetralogy of Fallot is a serious congenital heart defect characterized by a ventricular septal defect, pulmonary stenosis, and problems with right ventricular obstruction. It is a congenital disease that can cause hypoxia throughout the body, especially as age increases, the symptoms tend to worsen. After the corrective surgery for Tetralogy of Fallot, it does not mean that the patient is completely normal. Their lifespan is still somewhat impacted. They will not be like patients with simpler congenital heart diseases such as only a ventricular septal defect or atrial septal defect, who can achieve a natural lifespan. For these individuals, their lifespan is still impacted to some extent. As they age post-surgery, some complications may arise, including sudden death, and some may require a second surgery.
Can I eat eggs if I have had a myocardial infarction?
Myocardial infarction is a very serious type of coronary heart disease. An important pathological basis of coronary heart disease is the formation of atherosclerotic plaques in the arteries, which eventually rupture, leading to thrombosis that blocks the coronary arteries and causes myocardial infarction. Therefore, arteriosclerosis and hyperlipidemia are its pathological bases, and eggs contain a high amount of cholesterol. Hence, one should not consume eggs in excess. Eating one egg per day should still be acceptable, but consuming a large number of eggs can lead to increased blood lipids, which is harmful for the treatment and prevention of coronary heart disease. It is important to control the amount of eggs consumed, which should not exceed one per day.
Can Tetralogy of Fallot receive oxygen therapy?
Tetralogy of Fallot is a congenital heart defect that includes abnormalities such as ventricular septal defects, pulmonary valve stenosis, and stenosis of the right ventricular outflow tract. Due to these congenital defects, the ejection of blood from the heart is affected, which in turn impacts the blood supply to various systems and organs throughout the body. The patient may exhibit symptoms of cyanosis and hypoxia, especially after physical activity, hence the use of supplemental oxygen is advisable. While oxygen therapy can temporarily alleviate these hypoxic symptoms, it does not address the underlying issue. Surgery is necessary for a definitive resolution.
How to rescue sudden cardiac death from cardiogenic heart disease?
Firstly, the rescue approach for cardiogenic sudden death is the same regardless of the cause, whether it is rheumatic heart disease, coronary heart disease, malignant arrhythmia, or any other reason. If cardiogenic sudden death occurs, the rescue method is the same, which is to urgently perform cardiopulmonary resuscitation (CPR). CPR includes chest compressions of at least 100 per minute, and these must be effectively carried out. Secondly, artificial respiration must be performed, at about fourteen to fifteen times per minute. These two measures are the primary measures in the rescue of cardiogenic sudden death caused by rheumatic heart disease.
Does Tetralogy of Fallot involve chromosomal abnormalities?
Tetralogy of Fallot is primarily a developmental disorder, generally unrelated to chromosomes, with no chromosomal abnormalities. The cause of the disease is still not very clear. If pregnant again and giving birth again, it generally does not recur. Therefore, couples undergoing chromosomal testing before pregnancy is a common practice. It is generally believed that the four malformations in Tetralogy of Fallot occur due to viral infections acquired by the fetus during pregnancy, the mother consuming alcohol, or the use of certain medications during pregnancy. Currently, these are considered related factors, and so far, no abnormalities have been found related to genetics or chromosomes.
Is sudden cardiac death hereditary?
The majority of sudden cardiac deaths are caused by acute and extensive myocardial infarction, with some caused by malignant arrhythmias. Coronary atherosclerotic heart disease has a genetic history. Patients with such a family history, especially those with early-onset heart disease family history—that is, a family history of heart disease before the age of 50—should be given high attention. Heart disease has a genetic predisposition, and since sudden cardiac death is a type of heart disease, it also has a genetic predisposition.
The difference between cardiogenic sudden death and cerebrogenic sudden death.
The concept of sudden cardiac death exists, but there doesn't seem to be an equivalent term for "sudden brain death." For instance, if a large cerebral hemorrhage occurs and the patient dies within a very short time, this is referred to as death due to cerebral hemorrhage, not sudden death. Sudden death is characterized by death occurring unexpectedly within just a few minutes. The main difference between these, I think, is time. Sudden cardiac death happens very quickly and unexpectedly, which is why it's termed sudden death. In the case of brain-related issues, such as a significant cerebral hemorrhage, especially in the brainstem, it can compress the vital centers, leading to rapid respiratory and circulatory failure, and eventually death. Compared to sudden cardiac death, there is a slight delay in brain-related deaths; they don't occur as swiftly, and I believe the major difference lies in the timing.
Is sudden cardiac death related to eating a large number of eggs?
Sudden cardiac death is mostly due to coronary artery atherosclerotic heart disease, where the patient suffers from a large myocardial infarction. This is directly related to atherosclerosis, rupture of atherosclerotic plaques, formation of thrombosis, and obstruction of the coronary arteries. Therefore, the foundation of this disease is primarily hyperlipidemia and arteriosclerosis. Eating a lot of eggs is certainly not beneficial for arteriosclerosis and hyperlipidemia. We believe that there is definitely a connection, but it is not the only factor. For example, high blood pressure, diabetes, smoking, drinking, and other poor lifestyle habits are all related to heart disease. Thus, eating eggs is just one of the risk factors.