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Luo Juan
Endocrinology
About me
Associate Chief Physician, Department of Endocrinology, Hunan People's Hospital, Master's degree. Youth Committee Member of the Hunan Diabetes Professional Committee. Member of the Women's Endocrine Disease Prevention and Treatment Professional Committee of the Hunan Female Physician Association.
Proficient in diseases
Diagnosis and treatment of diabetes, thyroid disease, and adrenal gland diseases.
Voices
Is hyperuricemia contagious?
Hyperuricemia is not contagious. Hyperuricemia refers to a condition where the concentration of uric acid in the blood exceeds the normal upper limit. Typically, for males and post-menopausal females, a blood uric acid concentration greater than 420 µmol/L, or pre-menopausal females with more than 348 µmol/L, is considered hyperuricemia. Most severe cases of hyperuricemia are due to deficiencies in uric acid metabolism enzymes and fall under the category of liver metabolism disorders. Moreover, mild to moderate hyperuricemia is often associated with obesity, type 2 diabetes, hyperlipidemia, hypertension, arteriosclerosis, and coronary heart disease, among others. It is a type of nucleic acid metabolic disease, and therefore, it is not contagious.
Causes of Hypoglycemia in Females
The causes of hypoglycemia in women can be related to various factors, including drug-induced, hepatic, pancreatic, non-pancreatic tumors, renal, endocrine issues, as well as excessive consumption or insufficient intake, among others. For women, apart from organic lesions, common causes include excessive hunger, intense exercise, or chronic diarrhea, which can lead to hypoglycemia. Some women experience hypoglycemia due to lactation or pregnancy without timely replenishment of energy. Additionally, a common condition seen in women is idiopathic functional hypoglycemia, often found in neurotic women, which typically presents with symptoms of hunger and sweating, usually with few physical signs. Characteristically, it involves early-occurring reactive hypoglycemia that generally resolves on its own in less than 30 minutes, with blood glucose tests showing slightly decreased levels and normal or slightly elevated insulin levels in the blood.
The causes of hyperuricemia
The causes of hyperuricemia can currently be divided into two categories: primary and secondary. The cause of most primary hyperuricemia is not yet clear. A few cases are due to enzyme deficiencies, which are related to either excessive production or reduced excretion of uric acid. There is also idiopathic hyperuricemia, such as familial kidney diseases that are associated with hyperuricemia, among others. Some secondary hyperuricemias, for example, are secondary to some congenital metabolic diseases, such as glycogen storage disease. It can also be due to certain systemic diseases, such as leukemia, multiple myeloma, lymphoma, polycythemia vera, hemolytic anemia, etc. Some cases are physiological increases, such as the intake of foods rich in purines, long-term eating and fasting states can also cause increased blood uric acid. Some medications can also lead to hyperuricemia, such as thiazide diuretics, furosemide, ethanol, immunosuppressants, aspirin, etc., which can also cause hyperuricemia. Therefore, the causes of hyperuricemia are diverse, and some causes are still unclear.
Can thyroid nodules be treated?
It depends on the situation. For some patients, we can evaluate whether thyroid nodules are accompanied by changes in thyroid function. If there is an overactive thyroid, antithyroid treatment is necessary. If there is underactive thyroid function, appropriate thyroid hormone supplementation is needed, and some patients' nodules can shrink. Additionally, for some thyroid nodules, if there are no accompanying changes in thyroid function, and the nodules are assessed as benign through ultrasound and other evaluations, no special treatment is generally needed. It is recommended to monitor thyroid ultrasound and function every six months to a year. Furthermore, if some thyroid nodules grow rapidly or there is a potential for cancerous changes, or if there are significant compressive symptoms, we can also perform a fine needle aspiration biopsy of the thyroid cells, or surgical treatment. Therefore, whether thyroid nodules can be treated also depends on the situation.
What is gestational diabetes?
Gestational diabetes, also known as pregnancy-induced diabetes, generally refers to a condition diagnosed during pregnancy where varying degrees of high blood sugar levels are detected for the first time. This includes some cases where glucose intolerance or diabetes was undiagnosed before the pregnancy. Most patients see their blood sugar levels return to normal after delivery, but regardless of whether the high blood sugar normalizes post-pregnancy, it is considered gestational diabetes. Currently, the diagnostic criteria for gestational diabetes include a 75-gram oral glucose tolerance test. If the fasting blood glucose level is greater than or equal to 5.1 mmol/L, the blood glucose level after one hour is greater than or equal to 10.0 mmol/L, and the blood glucose level after 120 minutes is greater than or equal to 8.5 mmol/L, then gestational diabetes can be diagnosed.
The causes of obesity
Obesity can be classified into primary and secondary obesity according to its causes. Primary obesity, also known as simple obesity, is the most common type among various forms of obesity. It is mainly caused by poor dietary habits such as overeating, especially excessive intake of fatty foods, and a sedentary lifestyle, rather than being secondary to other diseases. Secondary obesity, on the other hand, is often due to diseases of the hypothalamic-pituitary axis, hypercortisolism, hypofunction of the thyroid or gonads, insulinomas, and other diseases. Secondary obesity is mainly due to diseases, and it can show some symptoms of the underlying disease.
Does hyperlipidemia cause dizziness?
Hyperlipidemia can cause dizziness. Hyperlipidemia involves the deposition of lipids on the endothelial lining of blood vessels, which is a primary consequence of abnormal blood lipids. This can lead to the onset and progression of atherosclerosis. For instance, low-density lipoproteins tend to deposit on arterial walls and are ingested by mononuclear giant cells. This situation can lead to arteriosclerosis, such as atherosclerosis of the cervical and intracranial arteries. This condition can result in insufficient blood supply to the brain, thereby causing dizziness. Therefore, hyperlipidemia can indeed cause dizziness.
Hyperuricemia treatment agent
Currently, the main treatments for hyperuricemia in clinical practice include the following types. One category is drugs that inhibit uric acid synthesis, primarily allopurinol and febuxostat. Allopurinol is a xanthine oxidase inhibitor, which mainly works by preventing the conversion of hypoxanthine and xanthine into uric acid through the inhibition of xanthine oxidase. Febuxostat, on the other hand, is a newer xanthine oxidase inhibitor and may be more effective than allopurinol in lowering blood uric acid levels. Another category includes drugs that promote the excretion of uric acid, suitable for patients with normal renal function and hyperuricemia. These mainly include probenecid, benzbromarone, and some use of thiazide diuretics, though their effectiveness for hyperuricemia is somewhat controversial. (Specific medications should be taken under the guidance of a physician.)
What fruits to eat for hyperuricemia?
For patients with hyperuricemia, we generally need to limit the intake of high-purine foods, such as crab and animal offal. Moderate consumption of medium-purine foods, such as meats and fish, is acceptable. Low-purine foods like vegetables, fruits, milk, and eggs can be safely consumed. For example, fruits like grapes, pears, grapefruits, kiwis, and dragon fruits are all acceptable to eat. However, many patients with hyperuricemia also have other metabolic abnormalities, such as high blood sugar. In such cases, it is important to monitor the quantity of fruit intake because excessive consumption can lead to fluctuations in blood sugar levels. Therefore, fruits can be safely consumed by those with hyperuricemia, but attention must be paid to any additional metabolic abnormalities that may be present.
Can people with hyperlipidemia eat peanuts?
People with high blood lipids can eat peanuts. Peanuts contain a rich amount of unsaturated fatty acids, which are beneficial to the human body in proper amounts. Firstly, they can regulate blood lipids, reduce some harmful cholesterol and triglycerides in the blood, effectively control the concentration of blood lipids, and can increase the content of beneficial high-density lipoproteins. Secondly, they can help clear blood clots, remove or alleviate the harm caused by animal fats from food, especially visceral fats, and prevent these lipid components from depositing on the walls of blood vessels to combat the formation and development of arteriosclerosis. Additionally, studies have shown that supplementing with DHA (an unsaturated fatty acid) can also enhance some of the body's immunity and improve self-immune function. Furthermore, it has some effects on improving arthritis or alleviating pain. Therefore, consuming peanuts in moderation is beneficial to the human body. However, peanuts are also high in calories, and excessive intake can lead to overweight or obesity, so they should not be consumed in excess and should be eaten in moderation.